About 30 percent of human lung cancer patients have mutations in Kras.
大约30%的人类肺癌病人有Kras突变。
When PIDD levels are artificially increased in human lung cancer cells, they become more resistant to cisplatin.
当PIDD水平在肺癌细胞人为增加时，它们对顺铂耐药性增强。
The cells they used included human lung cancer cells, human fibroblasts, which make up connective tissue, and mouse immune cells.
这些细胞包括人类肺癌细胞、成纤维细胞(构成结缔组织的)以及小鼠的免疫细胞。
Objective To establish a multidrug resistance cell line from human lung adenocarcinoma.
目的建立人肺腺癌多药耐药细胞系。
The studies indicated that the SOX4 gene may be involved in the human lung carcinogenesis.
研究表明SOX4基因的变化与肺癌的发生具有一定的关系。
RON variants can also express in human lung adenocarcinomas but not in squamous carcinomas.
肺腺癌组织中存在RON的变异体的表达，而鳞癌则没有。
These particulates are small enough to penetrate human lung tissue, causing severe health problems.
这些颗粒足够小，能进入人的肺组织，引发严重的健康问题。
Conclusion Specific and high titer antibodies against human lung cancer were successfully prepared.
结论成功制备高效且特异的抗人肺癌单克隆抗体。
Objective To investigate the telomerase activity in human lung cancer and its clinical significance.
目的研究肺癌组织中端粒酶活性的表达及其临床意义。
AIM: To investigate the gene expression profile of human lung squamous cell carcinoma in early stage.
目的：研究人早期肺鳞癌发生相关基因表达谱，探讨肺鳞癌发生的分子机制。
The intracellular pattern recognition receptor NOD1 is detected in human lung epithelial cells by RT-PCR.
并用RT-PCR的方法检测肺上皮细胞胞内模式识别受体NOD1的表达。
Objective: To investigate the effect and mechanism of insulin on human lung adenocarcinoma cell strain A549.
目的：探讨用胰岛素提高人肺腺癌细胞A549生长代谢水平及其机制。
There is one carcinogen in particular that researchers suspected for years contributed to human lung cancer.
多年以来研究人员猜测，有一种特别的致癌物质是导致人类得肺癌的罪魁祸首。
Conclusion The positive regulatory fragment of NHE 1 gene from human lung cancer cells was successfully cloned.
结论本实验已成功地克隆了人肺癌细胞NHE1基因调控序列中正调控序列片段。
Objective: to investigate the correlation between XRCC1 genetic polymorphisms and susceptibility to human lung cancer.
目的：探讨XRCC1基因单核苷酸多态性与肺癌易感性的关系。
Results There were obvious differences in gene expression between human lung cancer cells before and after ATRA treatment.
结果在全反式维甲酸诱导前后的细胞之间存在明显的基因表达差异。
Objective To investigate the effects of E1A gene on the proliferation rate and the cell cycle of human lung adenoma cell line.
目的探讨E1A基因对人肺腺癌细胞增殖的抑制作用与细胞周期的关系及作用机制。
Objective To study the inhibitory effect of isorhmnetin on human lung tumor YTLMC-90 cells and to elucidate its molecular mechanisms.
目的研究异鼠李素对人肺癌YTLMC-90细胞的抑制作用并探讨其作用机制。
Objective: to study the effect of the expression levels of Chemokine receptor CXCR4 on the metastatic potential of human lung cancer.
目的：探讨趋化因子受体CXCR4表达水平对人肺癌细胞转移潜能的影响。
The technique was found to perform better than either drug in isolation when tested in a mouse model targeting a human lung cancer tumor.
当针对人类肺癌肿瘤在小鼠模型中测试时，这项技术所呈现的效果比使用任一单一药物更好。
Objective: To determine the reversion of MDR of human lung carcinoma by ADM liposomes and ADM immunoliposomes modified by MRK-16 in vitro.
目的：制备MRK - 16修饰免疫脂质体，并检测免疫脂质体对非小细胞肺癌多药耐药细胞的杀伤及多药耐药逆转作用。
Objective to study the DNA damage and repair of normal lung interstitial cells and human lung adenocarcinoma cells exposed to cigarette smoke.
目的探讨经香烟烟雾溶液染毒的人正常肺间质细胞和人肺腺癌细胞的DNA损伤及其修复效应。
Objective To study the effect of GSSG modified superparamagnetic particles on the growth of human lung adenocarcinoma cell line SPC-A1 in vitro.
目的研究氧化型谷胱甘肽(GSSG)修饰的超顺磁性纳米粒子在体外对人肺腺癌细胞SPC - A 1生长的影响。
To screen differential expression protein of human lung adenocarcinoma cell A549 treated with radiation and to discuss the role of these proteins.
筛选了人肺腺癌细胞A549放射前后的差异蛋白质，并探讨了其在放射中的作用。
AIM: To study the effects of egtazic acid (EA) and calcimycin (Cal) on the synthesis of DNA and collagen in cultured human lung fibroblasts (HLF).
目的：研究依他酸（EA）和卡西霉素（Cal）对人胚肺成纤维细胞脱氧核糖核酸和胶原合成的影响。
The aim of this study is to establish a multidrug resistance cell line of human lung adenocarcinoma and study the mechanism of multidrug resistance.
本研究旨在建立人肺癌多药耐药细胞系，研究其耐药特性和机制。
Conclusion: Transfection of DTA gene in vitro may kill human lung adenocarcinoma cells and reduce the oncogenicity of human lung adenocarcinoma cells.
结论体外转染白喉毒素a基因可杀伤人肺腺癌细胞并降低人肺腺癌细胞的致瘤性。
Conclusion: Transfection of DTA gene in vitro may kill human lung adenocarcinoma cells and reduce the oncogenicity of human lung adenocarcinoma cells.
结论体外转染白喉毒素a基因可杀伤人肺腺癌细胞并降低人肺腺癌细胞的致瘤性。
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