PN with fat emulsion might be more rational than that with high glucose.
含脂肪乳剂的营养支持可能较高糖能源合理。
High glucose enhanced the expression of CTGF and FN, and insulin strengthened this effect.
高糖促进CTGF和FN的表达，胰岛素加强高糖此作用。
Aim To construct a nonalcoholic fatty liver model by feeding rats with high glucose and high fat diet.
目的用高糖高脂肪饲料喂养大鼠，建立大鼠非酒精性脂肪肝模型。
TRAIL-R4 was elevated in a time course manner under high glucose level in human aorta smooth muscle cells.
高糖对人主动脉平滑肌细胞TRAIL-R4表达上调的影响随着刺激时间的增加而增加。
Results Insulin enhanced the expression of GLUT4. High glucose decreased the expression of GLUT4 obviously.
结果胰岛素可增加GLUT4的表达，高糖使GLUT4表达明显下降。
Therefore we conclude that TGP could protect mesangial cells against oxidative stress induced by high glucose.
因此TGP可以保护高糖诱导的肾小球系膜细胞氧化应激损伤。
Conclusion: in vitro high glucose solution increased mesangial cell synthesis of extracellular matrix proteins.
结论：体外高糖可增加细胞外基质蛋白的合成。
Hypertension syndrome during pregnancy and macrosomia have been increased in women with gestational high glucose.
妊娠高血糖孕妇加大了发生妊高症、巨大儿的危险性。
Conclusions: Hypertension and insulin resistance can be induced in rats by feeding high glucose and high salt diet.
结论：高糖高盐饲料喂养大鼠能产生高血压及胰岛素抵抗。
AIM: to investigate the effect of high glucose with various concentration on apoptosis of human lens epithelial cells.
目的：研究不同浓度的高糖对人晶状体上皮细胞凋亡的作用。
Conclusion In Vitro, high glucose suppresses human fibroblast cell proliferation and stimulates PAI-1 gene expression.
结论高糖可抑制成纤维细胞增殖，刺激PAI- 1的表达。
AIM: To study the expression of FOXO1A in bovine retinal microvascular pericytes cultured with the high glucose medium.
目的：探讨高糖培养牛视网膜微血管周细胞FOXO1A表达。
You have to be careful because with high glucose you can develop gestational diabetes and it can harm both you and the baby.
你要小心，因为高糖可以开发妊娠糖尿病，它可以伤害你和宝宝。
Our brain needs glucose to function, and a high glucose meal will aid mental capacity considerably compared to a fasted brain.
大脑工作时需要葡萄糖，而与未进食的大脑相比，高糖饮食可以大大提升大脑的思维能力。
Conclusion Rosiglitazone can correct the endothelial cell dysfunction induced by high glucose and protect the vascular function.
结论罗格列酮可纠正高糖诱导的内皮细胞功能紊乱，起到保护血管的作用。
Conclusion High glucose can stimulate the proliferation of retina cells, the mechanism maybe lies in decreasing the concentration of NO.
结论高浓度葡萄糖可使视网膜混合培养细胞增殖，其机制可能部分与NO产生量减少有关。
AIM: to investigate the effect of high glucose on hydroperoxide (H2O2) -induced apoptosis in cultured bovine aortic endothelial cells (BAEC).
目的：研究高糖对过氧化氢(H_2O_2)诱导牛主动脉内皮细胞(BAEC)凋亡作用。
Conclusion: L-carnitine of certain concentration can promote the proliferation and inhibit the apoptosis of HPMC under high glucose condition.
结论：高糖条件下一定浓度的左旋肉碱可以促进人腹膜间皮细胞增殖、减少其凋亡。
Phosphorylation of IRS-1 at Ser612 by MAPK downregulates insulin signaling and may be part of a response to high glucose/glucosamine levels (11).
1的612位丝氨酸被MAPK磷酸化后会下调胰岛素信号，这可能是对高葡萄糖/葡萄糖胺比值的一种反应。
Objective: To investigate the effects of high glucose and heparin on the proliferation and expression of MMP2, MMP9 in normal human mesangial cells (NHMC).
目的：研究高糖对正常人类系膜细胞（NHMC）增殖及金属蛋白酶2（MMP2）和金属蛋白酶9（MMP9）表达的影响及肝素对其的干预作用。
Objective: To observe the effect of valsartan and low molecular weight heparin on the proliferation of glomeruli mesangial cells stimulated by high glucose.
目的：探讨缬沙坦和低分子量肝素对高糖刺激大鼠系膜细胞增殖的影响。
Objective To observe the effect of bradykinin(BK) on kidney mesangial cell multiplication and extracellular matrix secretion under high glucose environment.
目的观察缓激肽（BK）对高糖浓度下肾小球系膜细胞增殖和细胞外基质分泌的影响。
Objective To evaluate the apoptosis effectiveness and the mechanism of combined use of high glucose with high palmitic acid on human vessel endothelial cells.
目的观察高葡萄糖和高软脂酸联合应用对人血管内皮细胞凋亡的作用效果，并探讨其机制。
Maybe you are afraid of high glucose, or worried you would not be able to stop after eating a little bit. Or it could be that you simply don't want anything to eat.
或许你害怕吃了之后引起高血糖，或许你怕只吃一点点后很爱吃自己控制不住量，或者仅是你此时不想吃任何东西。
Conclusion Rosiglitazone can adjust the mRNA expressions of MMP-1 and TIMP-1 and inhibit the cumulation of extracellular matrix in kidney fibroblasts induced by high glucose.
结论罗格列酮可以通过调节MMP - 1和TIMP - 1的基因表达，抑制高糖导致的肾脏成纤维细胞外基质的蓄积。
AIM: To investigate the effect of high glucose on the expression of an endoplasmic reticulum stress marker glucose-regulated protein 78 (GRP78), and explore its underlying mechanism.
目的：观察高糖慢性处理对血管内皮细胞内质网应激标志蛋白葡萄糖调节蛋白78 (GRP78)表达的影响及其作用机制。
Conclusion: Fenofibrate can inhibit high glucose-induced proliferation of mesangial cells, decrease synthesis of extracellular matrix, and increase degradation of extracellular matrix.
结论：非诺贝特能抑制高糖培养的系膜细胞增殖，减少胞外基质合成，增加胞外基质的降解。
We investigated the cellular effect of high glucose using 3t3-l1 adipocytes on glucose transport activity, the expression of insulin signaling proteins and IRS1 tyrosine phosphorylation.
观察3t 3 -L1脂肪细胞长期暴露到高浓度葡萄糖对糖的转运活动和胰岛素信号蛋白的表达及磷酸化的影响。
Objective To study the effects of high glucose on activity of mitogen activated protein kinase (MAPK) in cultured human mesangial cells and to explain the mechanism of diabetic nephropathy.
目的：研究高糖对人肾小球系膜细胞丝裂原蛋白激酶（MAPK）活性的影响，探讨其在糖尿病肾病发病中的作用。
Objective To study the effects of high glucose on activity of mitogen activated protein kinase (MAPK) in cultured human mesangial cells and to explain the mechanism of diabetic nephropathy.
目的：研究高糖对人肾小球系膜细胞丝裂原蛋白激酶（MAPK）活性的影响，探讨其在糖尿病肾病发病中的作用。
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