Overexpressed FAT10 enhances the apoptosis in the starved HEK293 cells.
FAT10过表达促进饥饿状态的HEK293细胞发生凋亡。
CONCLUSION Cd-induced apoptosis involves mitochondrial pathway in HEK293 cells.
结论镉诱导HEK293细胞的凋亡可能通过线粒体途径。
Laser confocal microscopy was employed to reveal the localization of MRP in HEK293 cell line.
利用激光共聚焦显微镜观察MRP在HEK293细胞中的定位。
The recombinant vector was transfected into HEK293 cells with liposome transfection reagents for transient expression.
脂质体法转染HEK293细胞，用倒置荧光显微镜检测、分析其在HEK293细胞表达及定位。
Immortalized human embryonic kidney cell line HEK293 and the human embryonic lung fibroblasts HELF were used as positive control and negative control, respectively.
同时以端粒酶阳性的永生化人胚肾成纤维细胞HEK293作为阳性对照，以端粒酶阴性的人胚肺成纤维细胞HELF作为阴性对照。
Immortalized human embryonic kidney cell line HEK293 and the human embryonic lung fibroblasts HELF were used as positive control and negative control, respectively.
同时以端粒酶阳性的永生化人胚肾成纤维细胞HEK293作为阳性对照，以端粒酶阴性的人胚肺成纤维细胞HELF作为阴性对照。
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