This article reviews the proteins and transduction pat hw ay in endotoxic action.
综述内毒素作用中有关的蛋白及其传导通路。
Effect of polydatin on phospholipase A2 in lung tissues in rats with endotoxic shock.
虎杖甙对内毒素休克大鼠肺组织中磷脂酶a2的影响。
Methods Endotoxic shock in rats was induced by intravenous injection of dead gramegative bacteria E.
方法首先使用埃氏大肠杆菌死菌液制备大鼠内毒素性休克模型；
Objective: to study the mechanism underlying rhubarb inflammatory mediator response in endotoxic shock.
目的：研究大黄对内毒素性休克大鼠炎性介质作用的机制。
Objective to explore the changes and organ diversity of vascular reactivity following endotoxic shock.
目的了解内毒素休克后血管反应性的变化及器官差异。
Objective: to observe the effects of MCI 154, a calcium sensitizer, on cardiac function after endotoxic shock.
目的：研究钙增敏剂mci - 154对内毒素休克家兔心功能的影响。
Rats were challenged by E. coli LPS to set up the model of endotoxic shock, AG group were treated by aminoguanidine.
用大肠杆菌内毒素（LPS）复制大鼠内毒素性休克模型，氨基胍治疗组采用氨基胍治疗。
Objective: To investigate the changes of gut mucosal immune function in rats with endotoxic shock and its significance.
目的：探讨内毒素休克后大鼠肠粘膜免疫功能的改变及意义。
Objective To explore the changes of vascular reactivity following endotoxic shock and its relation with hemodynamic changes.
目的了解内毒素休克后血管反应性的变化规律及其与血流动力学变化的关系。
Objective: to investigate the effects of MCI 154, a calcium sensitizer, on myocardial calcium homeostasis during endotoxic shock in rats.
目的：研究钙增敏剂mci - 154对内毒素休克大鼠心肌钙稳态的影响。
CONCLUSION: CCK-8 improved the vascular reactivities in the presence of LPS, which may be one of the anti-endotoxic shock mechanisms of CCK.
结论：CCK-8能减轻LPS诱导兔离体胸主动脉反应性的异常改变，这可能是CCK-8抗内毒素休克作用的机制之一。
Conclusion: Calcium sensitizer can reverse the reduced responsiveness of myocardium to Ca2 + and improve myocardial contractility after endotoxic shock.
结论：钙增敏剂可逆转内毒素休克所致的心肌收缩蛋白对钙反应性的降低，改善内毒素休克机体的心肌收缩性能。
Method: Take Bacillus Coli endotoxin to copy rats endotoxic heat - stasis syndrome model, then test the effect of PRC on rheology and coagulation indexes.
方法：采用大肠杆菌内毒素复制大鼠内毒性热瘀证模型基础上，检测桃核承气汤对血液流变学和凝血指标的影响。
Conclusion There is organ diversity of vascular reactivity following endotoxic shock, which may be related to the changes of hemodynamics and blood redistribution.
结论内毒素休克血管反应性呈现一定器官差异，这种差异可能与内毒素休克血流动力学改变及血液重分布有关。
Methods The concentration of serum of ET and TNF of 20 critically ill patients, 18 non-critically ill patients, 10 rats with endotoxic shock and 10 normal rats were tested.
方法 我们检测了20例危重儿，18例非危重儿，10例内毒素性休克大鼠与正常大鼠的血浆内皮素与肿瘤坏死因子的含量。
The effects of chloroquine and dexamethasone, inhibitors to phospholipase A2(PLA2), on the activity of blood PLA2 and its related lipid mediators were observed in rabbits with endotoxic shock.
通过家兔内毒素休克模型重点观察了磷脂酶A2（PLA2）抑制剂磷酸氯喹和地塞米松对血中PLA2及其相关脂介质的影响。
Objective: to investigate the effects of MCI 154, a calcium sensitizer, on organ blood flows, arterial blood gases, some plasma biochemical parameters and survival rate in endotoxic shock rabbits.
目的：观察钙增敏剂mci - 154对内毒素休克家兔器官血流量、血气、某些血生化指标及动物存活率的影响。
Objective: to investigate the effects of MCI 154, a calcium sensitizer, on organ blood flows, arterial blood gases, some plasma biochemical parameters and survival rate in endotoxic shock rabbits.
目的：观察钙增敏剂mci - 154对内毒素休克家兔器官血流量、血气、某些血生化指标及动物存活率的影响。
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