Enard found that in the basal ganglia and connected regions involved in learning, the human version of FOXP2 caused some neurons to develop longer branches than those found in normal mice.
恩纳德发现在基底神经节和周围与学习有关的区域中，人类FOXP2基因导致有些神经元生长出比普通老鼠更长的突触。
Enard found that in the basal ganglia and connected regions involved in learning, the human version of FOXP2 caused some neurons to develop longer branches than those found in normal mice.
恩纳德发现在基底神经节和周围与学习有关的区域中，人类FOXP2基因导致有些神经元生长出比普通老鼠更长的突触。
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