CONCLUSION: Inhibition of KV function, increase in excitability and more positive Em in AM from COPD rats may be involved in the AM contribution to the COPD development.
结论：COPD大鼠肺泡AM细胞KV功能下调，细胞膜电位负值降低，兴奋性升高。该机制可能与AM细胞促进COPD发病的形成机制有关。
CONCLUSION: Inhibition of KV function, increase in excitability and more positive Em in AM from COPD rats may be involved in the AM contribution to the COPD development.
结论：COPD大鼠肺泡AM细胞KV功能下调，细胞膜电位负值降低，兴奋性升高。该机制可能与AM细胞促进COPD发病的形成机制有关。
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