It was found that the occurrence of epilepsy was not in close relation with the severity of brain hypoxia.
结果表明癫痫的有无与脑缺氧的严重程度并不成正比。
Preterm newborn child suffering from epilepsy in a higher proportion of perinatal asphyxia, chronic functional brain hypoxia, congenital dysplasia, and so is the cause of cerebral ischemia.
新生儿中早产儿患上癫痫病的比例较高，围产期窒息、慢性功能性脑缺氧、先天性脑发育不良、脑缺血等是致病原因。
At such heights, most people are susceptible to hypoxia, in which too little oxygen reaches body tissues, potentially leading to fatal lung or brain inflammation.
在这样的高地上，大多数人都容易受到缺氧的影响，因为在那里只有很少的氧气能输送到人体的各个组织，这样就可能导致致命的肺部或脑部的炎症。
Bleeding can cause foetal hypoxia (lack of oxygen to the brain of an unborn child).
出血会导致胎儿脑缺氧。
Hypoxia ischemic brain damage (HIBD), which resulted from neonatal asphyxia, is the main cause of death and disability in neonatal periods.
围产期窒息所致缺氧缺血性脑损伤(HIBD)是新生儿死亡和残疾的主要原因。
Objective to explore the roles of muscarinic acetylcholine receptor (m receptor) in rat brain injuries after soman intoxication combined with hypoxia.
目的探讨毒蕈碱样乙酰胆碱受体(M受体)在缺氧复合梭曼中毒脑损伤中的作用。
Objective To study the effect of intrauterine hypoxia on neonatal neurological behavior and brain responsive function.
目的了解宫内缺氧对新生儿神经行为和脑反应性功能的影响。
Conclusions the structural abnormalities of the hippocampal neuronal synapses is the morphological basis of abnormalities of higher brain functions of learning and memory induced by hypobaric hypoxia.
结论海马神经元突触结构异常是低压低氧条件下大鼠学习记忆脑高级功能障碍的形态学基础。
Hypoxic ischemic (HIE) of newborn is the damage in brain because of perinatal hypoxia or asphyxiation.
新生儿缺氧缺血性脑病(HIE)是指围产期缺氧窒息，导致脑的缺氧缺血性损伤。
Objective To discuss the effects and neuromechanism of hypobaric hypoxia on higher brain functions of learning and memory in rats.
目的探讨高原低氧对大鼠学习记忆脑高级功能的影响及其神经机制。
AIM: To observe the protective effects and mechanisms of ginseng and EGb mixture on hypoxia brain damage under simulated plateau environment condition in rats.
目的：观察人参银杏合剂对模拟高原缺氧脑损伤的保护作用并探讨其机理。
CONCLUSION: the gene expression of STAT3 and 5 in the hippocampal neurons reinforced by hypoxia plays an important role in brain injury caused by hypoxia.
结论：低氧增强海马神经元细胞中STAT 3,5基因的表达，在低氧所致的脑损伤中可能发挥重要作用。
BACKGROUND: brain injury often causes secondary cerebral ischemia and hypoxia, which aggravate the brain damages.
背景：脑损伤后常引起继发性脑缺血、缺氧，使脑损害加重。
Effects of Pretreatment with HBO on Brain SOD Activity and MDA Content following Rats Hypoxia-Ischemia in Uterus.
高压氧预处理对宫内缺氧缺血大鼠脑组织SOD活性、MDA含量的影响。
Epilepsy daily said that "epilepsy crazy," the main cause of the brain function of hypoxia, transient blurred.
癫痫病即人们日常说的“羊癫疯”，病因主要是脑部功能性缺氧、瞬间迷离。
Experts say people should know about other health concerns that can strike when traveling by air. One of these is hypoxia. It results from a lack of oxygen to the brain.
专家们说人们应该了解在乘飞机旅行时其他能够侵袭身体健康的问题，其中一个是组织缺氧，它是由大脑缺氧造成。
Conclusions: The brain oxygen metabolism monitoring is a safe and reliable method to detect brain tissue hypoxia and acidosis after SHI.
结论：脑氧代谢持续监测安全、有效，有利于早期发现SHI后脑组织缺氧及酸中毒。
Generally outside the serious injury, particularly brain injury caused by hypoxia, the greater the possibility of occurrence of juvenile epilepsy.
一般外伤愈严重，特别是外伤引起脑部缺氧者，发生青少年癫痫的可能性愈大。
Conclusion Propofol may inhibit hypoxia in the brain and the apoptosis of nerve cells in result of protecting the cerebral ischemia and reperfusion damage in rats.
结论异丙酚可能通过抑制大脑缺氧，抑制神经元的凋亡，从而对大鼠局灶性脑缺血-再灌注损伤发挥保护作用。
AIM: to explore the characteristics of energy metabolism in brain mitochondria of rats exposed to acute and chronic hypoxia.
目的：探讨缺氧大鼠脑线粒体能量代谢的特点。
Results Nitric oxide was directly toxic to PC12 cells in a dose-dependent manner, and brain tissue homogenate extracts taken from hypoxia preconditioned mice could prevent PC12 cells from NO toxicity.
结果NO对PC12细胞可以产生剂量依赖性的直接毒性作用，而缺氧预适应小鼠脑匀浆提取液能有效对抗NO神经毒性作用。
The content of lipofuscin(LF) estimated for brain and heart of mice. The stress power was observed. The ability of anti tired and tolerance hypoxia and phago-function of macrophage were estimated.
脑、心脂褐质（LF）的含量和小鼠应激能力：抗疲劳和耐缺氧实验及小鼠腹腔巨噬细胞吞噬功能测定。
Water soluble antihypoxic or hypoxia-resistant elements might exist in the brain of hypoxia resistant animals, which were extractable, transferable, and permeable to the blood brain barrier.
急性重复缺氧小鼠脑中的一种或多种可提取并可通过血脑屏障的水溶性物质可能为该适应变化的一部分。
High sustained + Gz acceleration produced by modern high performance aircrafts can induce brain damage because of ischemia and hypoxia.
现代高性能战斗机产生的高g值持续性正加速度可导致脑缺血、缺氧，继而引起脑功能障碍。
Conclusions When the brain is severely injured, there is cerebral hypoxia or ischemia during the first 24 hours, and cerebral hyperemia in the following second-fourth days.
结论重型脑损伤后24小时内存在脑缺氧、缺血，伤后第2 - 4天为脑氧合过度、脑充血。
Impairment of neuronal mitochondria following hypoxia of brain not only result in nerve cell's energy-deprivation and dysfunction, mitochondria also play key roles in apoptosis of neurons.
脑缺氧后神经元线粒体损伤不单使细胞发生能量缺失和功能丧失，还可以介导凋亡调节信号，是缺氧损伤后神经元凋亡的一个中心环节。
RESULTS: All the 148 cases of hypoxia-ischemia brain injury were involved in the result analysis.
结果纳入缺氧缺血性脑损伤患儿148例，全部进入结果分析，无脱落。
Conclusion It is suggested that, the increase of ET and NO contents of cerebral cortex and thalamus may participate in the pathogenesis of hypoxia brain injury in old rats.
结论急性低压缺氧可引起老龄大鼠大脑皮质和丘脑中et、NO含量明显升高以及大脑皮质血管内皮细胞和神经细胞明显损伤，ET、NO可能参与了缺氧性脑损害的病理过程。
Conclusion It is suggested that, the increase of ET and NO contents of cerebral cortex and thalamus may participate in the pathogenesis of hypoxia brain injury in old rats.
结论急性低压缺氧可引起老龄大鼠大脑皮质和丘脑中et、NO含量明显升高以及大脑皮质血管内皮细胞和神经细胞明显损伤，ET、NO可能参与了缺氧性脑损害的病理过程。
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