Free radicals, linked to rapid aging, are highly reactive with other molecules, including vital DNA and proteins, the destruction of which can damage or kill cells.
与迅速衰老相关联的自由基很容易与包括维持生命所必需的DNA和蛋白质发生反应，而破坏这些DNA和蛋白质，就可能会损伤或杀死细胞。
DNA repair is essential for the accurate preservation of genetic information and to ensure the healthy functioning of cells, and a connection between aging and DNA damage has long been suspected.
DNA修复对遗传信息的准确保存和确保细胞的正常功能是必需的，年龄和DNA损伤的关系一直存在疑问。
The relation between mutation and DNA damage and cell aging is discussed in this paper.
对降低基因突变和DNA损伤与细胞老化的关系作了讨论。
Objective to observe the effects of zinc-deficiency (ZD) on antioxidation system and repair of DNA damage and of hepatic cells by establishing an aging mice model induced with D-galactose.
目的通过d半乳糖诱导小鼠衰老模型，探讨缺锌对衰老小鼠抗氧化系统和肝脏dna损伤与修复的影响。
These molecules can react with cells, membranes and DNA, causing skin damage and signs of aging.
这些分子能与细胞，隔膜和DNA起反应，导致皮肤受损并显现衰老迹象。
Deacetylation of p53 occurs through interaction with the SIRT1 protein, a deacetylase that may be involved in cellular aging and the DNA damage response (19).
SIRT1蛋白，一种涉及到细胞衰老和DNA损伤反应的去乙酰基酶能够与p53结合，促使后者的去乙酰化（19）。
Deacetylation of p53 occurs through interaction with the SIRT1 protein, a deacetylase that may be involved in cellular aging and the DNA damage response (19).
SIRT1蛋白，一种涉及到细胞衰老和DNA损伤反应的去乙酰基酶能够与p53结合，促使后者的去乙酰化（19）。
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