• The plasma ADMA levels are elevated in EH patients.

    ②高血压病患者血浆ADMA的水平升高;

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  • There is abundant evidence that ADMA may be a novel risk factor for cardiovascular disease.

    大量研究证明,ADMA可能是心血管疾病的一个新的危险因子。

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  • Fenofibrate reversed the effect of ADMA on the expression of MMP-9 and promoted the synthesis of no.

    非诺贝特可以抑制adma诱导人脐静脉内皮细胞所致MMP - 9的表达增加并促进NO的合成。

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  • Conclusions: Plasma ADMA levels predict fatal and non-fatal cardiovascular events in patients with type 1 nephropathy.

    结论:在1型糖尿病肾病患者中,血adma的水平对致命和非致命的心血管事件有预测作用。

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  • The ADMA plasma level also tended to increase with an increasing number of classic cardiovascular risk factors, the researchers note.

    研究者还注意到:adma血浆水平倾向于随着传统的心血管风险因子种类的增加而增加。

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  • This article reviews the effects of ADMA on endothelial function and its mechanism and the research into related therapeutic procedures.

    现就adma对内皮功能的影响和机制以及相关的治疗研究现状进行综述。

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  • These results suggest that in the older group of rats, the elevated level of ADMA may be a contributor to vascular endothelial dysfunction.

    提示老龄大鼠血中二甲基精氨酸浓度升高可能是促进血管内皮功能不全的重要原因。

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  • CONCLUSION: Endothelial dysfunction induced by OFR is associated with the increase in ADMA concentration and reduction of DDAH activity, but not DDAH expression.

    结论:OF R培养下,内皮损伤adma的增加与DDAH的活性减弱有关,而与DDAH的表达无关。

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  • Objective to explore the role of endogenous nitric oxide synthase inhibitor asymmetric dimethylarginine (ADMA) in different factors-induced gastric mucosal injury.

    目的:研究一氧化氮合酶抑制物非对称性二甲基精氨酸(adma)在不同因素诱发的胃黏膜损伤中的作用,并初步探讨其机制。

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  • In the overall population, ADMA plasma levels correlated with body-mass index, creatinine levels, and plasma triglyceride levels, but not with age, the results indicate.

    在全部人群中,ADMA血浆水平与体重指数、肌氨酸酐水平和血浆甘油三酸酯水平相关,但与年龄、结果显示不相关。

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  • Conclusion taurine protects the endothelium against damages induced by LDL, and the protective effect of taurine on endothelium is related to the reduction of ADMA concentration in rats.

    结论牛磺酸对LDL诱导的血管内皮细胞损伤有保护作用,其保护作用与降低adma浓度有关。

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  • CONCLUSIONS: OFR induces endothelial dysfunction through increasing ADMA concentration, while captopril relieves endothelial dysfunction induced by ox-LDL through decreasing ADMA concentration.

    结论:OFR通过增加ADMA导致内皮功能紊乱,卡托普利则能通过减少ADMA减轻OFR诱导的内皮细胞代谢功能障碍。

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  • ObjectTo study the effect of polydatin (PD) and asymmetric dimethylarginine (ADMA) on the endothelial function in aorta vascular strips of healthy rabbits and the interaction between PD and ADMA.

    目的研究虎杖苷(PD)与非对称性二甲基精氨酸(adma)对正常兔主动脉血管条内皮功能的影响及PD与adma之间的相互作用。

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  • In the present study, the blood content of calcitonin gene-related peptide (CGRP) and asymmetric dimethylarginine (ADMA) in 6 months old rats was detected in comparison with that of 20 months of rats.

    检测6月龄和20月龄大鼠血中二甲基精氨酸和降钙素基因相关肽浓度的变化。

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  • In the present study, the blood content of calcitonin gene-related peptide (CGRP) and asymmetric dimethylarginine (ADMA) in 6 months old rats was detected in comparison with that of 20 months of rats.

    检测6月龄和20月龄大鼠血中二甲基精氨酸和降钙素基因相关肽浓度的变化。

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