Progesterone enhances the ability of octreotide to inhibit the growth of A549 cells.
孕酮联合奥曲肽对A549细胞的生长具有更强的抑制作用。
Methods A549 cells cultured in vitro were divided into heat stress group and BPDE group.
方法体外培养的A549细胞分为高温应激组和BPDE组。
The results showed that 1 cause a persistent increase in cytoplasmic calcium levels in A549 cells.
结果显示1具有促进钙离子细胞内流的活性。
In conclusion, our data demonstrated that:MP infection may affect protein expression in A549 cells.
通过上述研究，我们得出以下结论：MP感染可引起A549细胞内蛋白表达的变化。
OBJECTIVE To investigate the optimum condition of polyethylenimine(PEI) for gene transfer to A549 cells.
目的考察并确定多聚乙烯亚胺（PEI）最佳的体外细胞转染条件，评价PEI作为基因转染试剂的可行性。
Objective To observe the effects of matrine and oxymatrine on the proliferation and the apoptosis of A549 cells.
目的观察比较苦参碱及氧化苦参碱对A5 49细胞系增殖能力的影响及其对A5 49细胞凋亡的诱导效应。
Objective To investigate the roles of PI3K pathway in multicellular resistance of lung adenocarcinoma A549 cells.
目的探讨PI3K信号通路在肿瘤群集耐药中的作用。
With incubating time prolonging, four organelles' ability to absorb HMME is enhancing in A549 cells, especially does lysosome;
随着孵育时间的延长，A549肺癌细胞各细胞器吸收HMME能力逐渐增强，尤以溶酶体显著；
Conclusion The combined use of RSG and CDDP can induce apoptosis of A549 cells and it shows evidently time and dose dependence.
结论罗格列酮与顺铂合用能诱导肺癌A549细胞调亡，呈明显的时间及剂量依赖性。
Methods the chloride channel currents were recorded in cultured A549 cells by using the whole-cell mode of patch clamp techniques.
方法在离体培养的A549细胞上，用膜片钳全细胞模式记录氯通道电流。
Objective:To compare the adverse effects of the gasoline exhaust and methanol exhaust on proliferation and apoptosis in A549 cells.
目的：比较甲醇燃料汽车尾气和汽油燃料汽车尾气对A549细胞株的细胞毒性及增殖和凋亡功能的影响。
Conclusion The overexpression of CC10 might play an important role in the matrine-induced apoptosis of lung adenocarcinoma A549 cells.
结论CC10基因表达增高可能在苦参碱诱导肺腺癌A549细胞凋亡中起重要作用。
Progesterone combined with octreotide was superior to progesterone or octreotide alone in inhibiting the growth of A549 cells(P<0.05).
孕酮联合奥曲肽抑制A549细胞生长的作用比奥曲肽单药更强（P<0.05）。
Objective to investigate the proliferation and apoptosis effect of Parthenolide on human non-small cell lung cancer A549 cells in Vitro.
目的研究小白菊内酯在体外对人非小细胞肺癌a 549细胞增殖及凋亡的作用。
METHODS: We used RT-PCR, flow cytometry and immunofluorescence cell staining techniques to observe the expression of PARs on A549 cells.
方法：采用逆转录聚合酶链反应(RT - PCR)、流式细胞仪和免疫荧光细胞染色分析技术分析肺上皮细胞系a549细胞PAR s的表达情况。
Results: The proliferation inhibitory rate of PDTC on A549 cells increased as incubation time and concentration increasing(P<0.05 or 0.01).
结果：随着PDTC干预浓度的增大和时间延长，A549细胞生长抑制率增加（P<0.05、0.01）；
Results Microwave obviously inhibited the activity and inducted the apoptosis of A549 cells. Time dependence and dose dependence were definite.
结果微波明显抑制了A549细胞的活力，并诱导A549细胞发生凋亡，辐射效果与辐照剂量、作用时间相关。
Conclusions S2-protein from SARS coronavirus inhibits the chloride channel function in A549 cells, and both PKC and P38 are not involved in mediating this modulation.
结论SARS冠状病毒S2蛋白能抑制A549细胞氯通道的功能活动，PKC和P38可能不参与S2蛋白对通道电流的抑制作用。
The formulated complexes exhibited a considerably higher transfection efficiency against FOL receptor over-expressing KB cells than FOL receptor deficient A549 cells.
与缺乏FOL受体的A549细胞相比，由此而来的合成物在过量表达FOL受体的KB细胞中有更高的转染率。
Conclusion Aspirin can inhibit the proliferation of lung adenocarcinoma cells A549 through influencing the distribution of cell cycle and inducing apoptosis of A549 cells.
结论阿司匹林可抑制肺腺癌细胞A549的增殖，其影响细胞周期分布、诱导细胞凋亡可能是其重要的机制。
AIM: to study the apoptosis of human lung adenocarcinoma A549 cells induced by CN-2, a derivative of podophyllotoxin, and to obtain insights into its molecular mechanism of action.
目的研究鬼臼素衍生物cn - 2对人肺腺癌细胞A549的凋亡诱导作用，并对其分子机制进行初步探讨。
A549 cells were pretreated with inhibitor of PLD(1- butanol), the invasion of A. fumigutas into host cells were detected to evaluate the role of PLD in A. fumigutas entry into host cells;
用不同浓度的PLD蛋白化学抑制剂（1-丁醇）预处理细胞，测定其对烟曲霉分生孢子侵染率的影响；
RESULTS All four extracts of traditional Chinese medicinal materials showed growth inhibition to breast cancer MCF7 cells while only sodium ferlate showed inhibition to lung cancer A549 cells.
流式细胞法测定细胞周期。结果4种中药对乳腺癌MCF7细胞的增殖均有不同程度的抑制作用，但只有川芎素可明显抑制肺癌A5 49细胞的增殖；
Western blot analysis of extracts from A549 cells, untreated or treated with calf intestinal phosphatase (CIP), using Phospho-FAK (Tyr397) (D20B1) Rabbit mAb (upper) and FAK Antibody #3285 (lower).
未处理或经过牛小肠碱性磷酸酶（CIP）处理的A549细胞提取物使用Phospho-FAK （Tyr397） （D20B1）兔单抗（上）和FAK抗体#3285（下）进行western blot分析。
Objective To investigate the chemosensitivity of irradiated resistant cells of multicellular spheroids in A549 lung adenocarcinoma.
目的探讨A549肺腺癌多细胞球体放射抗拒细胞的化疗药物敏感性。
These findings suggest that BSO can effectively overcome the drug resistance of A549/R2 through its cytotoxic effects on the cells.
结果表明：BSO能有效地逆转A549／R2细胞的抗药性或对其直接产生细胞毒作用；
Rosiglitazone and carboplatin in combination inhibited tumour growth in xenograft models using A549 NSCLC cells and ES2 ovarian cancer cells, without significant toxicity.
用A549NSCLC细胞和ES2卵巢癌细胞建立异体移植模型，罗格列酮和卡铂共同抑制肿瘤的生长并且没有明显的副作用。
Objective To study the inhibition of lung cancer cells of primary culture and A549 cell line on the proliferation of tumor-infiltrating lymphocytes (TIL) in vitro and its clinical implications.
目的研究肺癌原代细胞和细胞系a549对肿瘤浸润性淋巴细胞的抑制作用及其临床意义。
To investigate the fraction of cancer initiating cells in lung cancer cell line A549 by cloning and tumorigenic analyses.
目的通过克隆形成及致瘤能力研究肺腺癌细胞系a549肿瘤始发细胞的比例。
To investigate the fraction of cancer initiating cells in lung cancer cell line A549 by cloning and tumorigenic analyses.
目的通过克隆形成及致瘤能力研究肺腺癌细胞系a549肿瘤始发细胞的比例。
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