本室已经建立了MHC不相合单份鼠胎血同种移植的小鼠模型。
We have constituted a mouse model for fetal blood transplantation histocompatibility complex (MHC) without causing serious GVHD.
被动吸烟可使鼠胎神经管上皮细胞过度凋亡，实验组均较对照组有明显增多的细胞凋亡。
Smoking can cause excessive apoptosis in neural tube of experimental group, more apoptosis cells were observed in neural epithelium compared with the control group.
孕早期、中期用药组母鼠重量、胚胎数减少，吸收点增多，但不影响鼠胎、胎盘的重量。
In the early and middle pregnancy the weight of pregnant mouse and the number of fetus of AG groups was decreased. The number of fetal resorptions was increased.
材料与方法：用组织块培养方法作SD大鼠胎鼠骨源性成骨细胞的体外培养，获取实验细胞模型并划分其分化阶段。
Materils and method: Obtaining experimental cellular model by isolating and culturing in vitro of osteoblasts from SD rats' fetal calvaria . Differentiation stages were divided .
斯坦福大学魏斯曼实验室最近向鼠胎注射进一些人神经干细胞，制造出的小鼠大脑细胞中，掺杂有约1%为人脑细胞。
David Waisman Laboratory at Stanford University recently injected into fetal mouse neural stem cells in some people, to create the mouse brain cells, about 1% of the well-doping brain cells.
从胎鼠中取出的肺细胞被注入这个装置系统中，同时还有帮助细胞生长的营养物质。
Lung cells taken from rat fetuses were injected into the setup, along with nutrients that helped the cells grow.
本文研究了敌草快和肾上腺皮质激素对胎鼠动脉管的收缩作用。
The toxic effects of diquat and adrenocortical hormone on ductus arteriosus of rat fetus were studied in this paper.
然后研究者把发育中的胎鼠的脑暴露于损伤。
Then the researchers exposed the brains of the developing fetuses to injury.
结论：孕酮对离体胎鼠头盖骨成骨细胞的分化具有多重促进效果，但对细胞的增殖无影响。
Conclusion: Progesterone has a multi stimulating effect on the differentiation of fetal rat calvarial osteoblast, but no effect on cell proliferation.
结论：胎鼠听囊细胞在体外自然培养条件下可以分化或者增殖为未成熟毛细胞。
CONCLUSION: in natural culture condition in vitro, otocyst cells of fetal rats can differentiate or proliferate to immature hair cells.
方法胎鼠头盖骨成骨细胞在含有不同浓度孕酮的培养基中培养约20天。
Methods Fetal rat calvarial osteoblasts were cultured in the medium with different concentrations of progesterone for nearly 20 days.
结论孕酮从基因水平调节离体胎鼠成骨细胞骨钙素的生成，这种调节呈现时间及剂量依赖。
Conclusion progesterone was involved directly in osteocalcin production by regulating gene expression and this effect of progesterone was time and dose dependent.
结论维甲酸有较强的胎鼠骨骼致畸作用，这种作用是多部位的。
Conclusion Retinoic acid has a strong and multi-regional teratogenic effect on the skeleton of fetal rat.
目的：探讨诊断超声对胎鼠内耳细胞是否构成刺激。
Objective: To evaluate whether diagnostic ultrasound should stimulate inner ear of fetal rats.
结论：FGR胎鼠在宫内即可有神经系统损伤，并对出生后远期的学习记忆能力产生影响。
Conclusion: FGR may induce the nervous system damage in the intrauterine fetal rats and further affect the learning and memory ability after birth.
结果：康乃精颗粒对大鼠的一般状况、体重变化和胎鼠发育、母鼠分娩、新生仔鼠以及子代大鼠的发育状况均无任何毒性作用。
Results: KNJ had no toxicologic effect in the general status, body weight change, and embryonic rat development, maternal rat delivery, newborn rat as well as the development status of rat.
目的了解蚂蚁粉对宫内发育迟缓（IU GR）胎鼠的大脑，肝脏和胎盘的作用。
Objective To investigate the effect of ant powder on the brain, liver and placenta of fetal rat with IUGR.
观察母鼠和胎鼠的生长发育情况。
目的探讨胎肝胰岛细胞联合移植治疗糖尿病大鼠的效果，以及胎肝细胞移植诱导糖尿病受者鼠免疫耐受的可行性。
Objective to study the effect of combined transplantation of pancreatic islets and hepatocytes on the treatment of diabetic rats and the possibility on hepatocytes inducing islet allograft tolerance.
结论：NOS抑制剂通过减轻缺血缺氧脑损伤时胎鼠脑神经细胞凋亡对其出生后的远期智能起保护作用。
Conclusion: NOS inhibitors play a obvious protective role in long-term intelligence of fetal rat after delivery through relieved neuron apoptosis during hypoxic ischemia encephalopathy.
期末血清叶酸、胎鼠血清叶酸及胎鼠脑组织DNA甲基化水平作相关分析。
Correlation analysis was processed among serum folate concentration of dam and fetus and DNA methylation level in fetal brain.
目的研究氟桂利嗪(FNZ)、拉莫三嗪(ltg)及两药联合经孕鼠给药，对胎鼠宫内缺血缺氧性脑损害的保护作用。
Objective To investigate the neuroprotective effects of flunarizine (FNZ) and lamotrigine (LTG), and their combination on the hypoxic-ischemic brain damage in fetal rats.
结果表明：胎鼠成纤维细胞MEF培养组的囊胚率和胚胎细胞数38。
The result showed that the mouse embryonic fibroblasts MEF group achieved significantly higher blastocyst formation rate 38.
目的探讨胰岛素样生长因子结合蛋白- 1 (IGFBP - 1)在正常胎鼠发育和胎鼠宫内发育迟滞(IUGR)发生中的作用。
Objective To explore the role of IGFBP-1 in the normal growth and intrauterine growth retardation (IUGR) in rat fetuses.
结论血清igfbp - 1作为生长抑制因子参与正常胎鼠发育及IUGR的发生。
Conclusions Serum IGFBP-1 participates in the growth of normal and IUGR rat fetuses as an inhibiting factor.
目的观察胎鼠发育过程中脑内神经干细胞增殖、迁移和分化特点。
Objective To evaluate the characteristics of proliferation, migration and differentiation of neural stem cells in the brain of rat embryo during development.
目的应用胎儿外科技术，建立胎鼠宫内先天性唇裂及无瘢痕愈合的伤口模型，并与成年鼠伤口愈合过程进行组织学对照观察，为进行唇裂宫内修复的研究提供依据。
Objective To properly evaluate the benefits of in utero repair of cleft lip, an animal model of cleft lip was developed and the fetal scarless wound healing was observed in SD rat fetus.
用骨骼染色法观察胎鼠有无骨骼畸形，内脏切片法观察胎鼠内脏畸形情况。
The fetus skeletal development and visceral abnormality were observed by skeletal staining and visceral section.
用骨骼染色法观察胎鼠有无骨骼畸形，内脏切片法观察胎鼠内脏畸形情况。
The fetus skeletal development and visceral abnormality were observed by skeletal staining and visceral section.
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