QT间期延长综合症会加速心跳，导致昏厥。
Long QT syndrome speeds up heartbeats, and can cause fainting.
先天性Q- T间期延长综合征是一组家族遗传性疾病。
Congenital long QT syndrome is a group of familial inherited disease.
问题的关键在于这种QT间期延长是与药物控制血糖的作用相关联的。
The problem is that it's correlated to the effect on the glycemic control which is well known to increase the QT.
这些细胞最特别之处在于，它们来自于一位遗传性QT间期延长综合症患者。
What's particularly special about these cells is that they came from a patient with one of the inherited forms of long QT syndrome.
现对导致QT间期延长的原因、QT间期的测量及相关治疗等方面进行综述。
Here is to review on the reasons of QT interval prolongation, the measurement of QT interval and related treatments.
当把神经调节蛋白给予哺乳动物时，发现其心电图pr间期延长，心率减慢。
When the neuregulin is given to a mammal, the electrocardiogram pr interval thereof is found to be prolonged, and the heart rate is slowed down.
不知为何，QT间期延长综合症患者可以许多年毫无症状，而后突然死于心停跳。
For unknown reasons, people with long QT syndrome can go without symptoms for years, and then suddenly drop dead from cardiac arrest.
目的探讨经电视胸腔镜切除左胸交感神经治疗先天性QT间期延长综合征的方法和效果。
Objective to assess the feasibility, safety and effectiveness of video-assisted thoracoscopic sympathectomy (VATS) for the treatment of congenital long QT syndrome.
工人接触后心电图指标与接触前自身比较，出现心率减慢、PR间期和QT间期延长及血压下降。
Their heart rates slowed down, P R and Q T intervals prolonged and blood pressure reduced after exposure, as compared with those before exposure.
根据美国心脏病学院(的统计)，QT间期延长的人心源性死亡的风险增高，其可解释美国每年超过四十万的死亡人数。
People with prolonged QT intervals have a higher risk of cardiac death, which accounts for more than 400,000 deaths each year in the United States, according to the American College of Cardiology.
本文通过二例及家系发病调查，对家族性q—t间期延长综合征的病因、发病机理、临床、心电图表现及治疗进行了讨论。
In this article are discussed the etiology, pathogenesis, Clinical and ECG manifestation as well as treatment of the familial long q-t interval syndrome through census of familial systems.
方法对39例PR间期延长(研究组)和141例PR间期正常(对照组)患儿房室传导功能进行了经食道心房调搏研究。
Methods The transesophageal pacing was performed in 39 children with prolonged pr interval (study group) and 141 children with normal pr interval (control group).
结果阿司咪唑使多数小鼠心率减慢，PR间期和Q T间期延长，随剂量增加，甚至可引起完全性房室传导阻滞和窦性停搏。
RESULTS astemizole could slower the heart rate of mice, prolong P-R and Q-T interphase, even arise the complete AV block and sinus beat stop with the dose increase.
窦性心动过缓、ST-T改变、心肌缺血最常见，其他异常主要有早搏、心律不齐、传导阻滞、异常Q波、Q-T 间期延长等改变。
The ECG abnormalities were commonly sinus bradycardia, ST-T changes, and myocardial ischemia while others mainly included premature beat, arrhythmia, conduction block, abnormal Q waves, and so on.
对实验牛心血管系统、呼吸系统，肝肾系统和血液指标监测，仅见心率减慢、轻度酸碱平衡紊乱以及ECG的P—R间期延长，其他指标均无明显变化。
In monitor of the chief systems of experimental cattle, only bradycadia, mild imbalance of acid-base and prolonged P-R interval of ECG were found.
氟代喹诺酮和大环内酯抗菌剂都与QT间期的延长有关。
Fluoroquinolones and macrolide antimicrobial agents have both been associated with prolongation of the QT interval.
项目参与的时间期限不允许被延长。
结果：随着触发间期的延长，心肌灌注缺损范围减小。
Results:The longer the trigger interval lengthened, the more the contrast deficit of risk areas decreased.
血清酒精浓度越高，ECG间期显著延长并可能出现潜在的传导紊乱或甚至SCD的发生率越高。
The higher the blood alcohol concentration, the higher the occurrence of a significant extension of ECG intervals with possible manifestation of latent conduction disturbance or even SCD.
通过比较心房扑动（简称房扑）成功消融前后右心房峡部时间间期，分析峡部时间间期的延长程度对峡部完全性双向阻滞的预测价值。
The purpose of this study was to determine whether the extent of prolongation of the transisthmus interval after ablation predicts complete bidirectional block.
通过比较心房扑动（简称房扑）成功消融前后右心房峡部时间间期，分析峡部时间间期的延长程度对峡部完全性双向阻滞的预测价值。
The purpose of this study was to determine whether the extent of prolongation of the transisthmus interval after ablation predicts complete bidirectional block.
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