结论：癫痫急性发作期n 1、P 1波的改变与兴奋性氨基酸的神经元毒性作用及继发性缺血、缺氧致神经元损伤有关。
Conclusion: the changes of N1P1 in acute attack period are related with the neuron toxic action of excitatory amino acids and lesions made by secondary ischemia and hypoxia.
增加胃溃疡的作用，阿司匹林药理下降，相反中断类固醇和高剂量使用阿司匹林会出现水杨酸的毒性。
Increased ulcerogenic effect; decreased pharmacologic effect of aspirin. Conversely, salicylate toxicity may occur in patients who discontinue steroids with concurrent high-dose aspirin therapy.
目的：探讨米酵菌酸对人膀胱癌细胞毒性、杀伤作用及其超微结构的影响。
Objective:To investigate the toxicity and effect of bongkrekic acid (BKA) on the ultrastructure of human bladder carcinoma cells cultured in vitro.
本研究的目的是探讨低毒性的食品调味剂桂皮酸对PGCL3人肺癌细胞的抑制作用。
This study is intended to investigate the anticancer effects of the low toxic flavorings cinnamic acid on PGCL3 human lung cancer cell subine.
结论黄芩苷对大鼠脑片缺氧缺糖性损伤具有一定的保护作用，作用机制可能与其抑制了兴奋性氨基酸毒性有关。
Conclusion Baicalin possesses protective effect on OGD injury in rat brain slices, which is related to its inhibitory effect on excitatory amino acids toxicity.
目的观察米酵菌酸对小鼠顶叶皮质毒性作用的超微结构改变，探讨米酵菌酸的中毒作用机理。
Objective To observe the ultrastructure changes of intoxicated mice parietal cortex caused by bongkrekic acid (BKA) and to explore the mechanism of BKA poisoning.
而二者分泌柠檬酸的速率虽然均有显著提高，但处理后感抗品种之间差异不大，表明柠檬酸在缓解铝毒性中的作用不大。
The rate of citrate exuded in the two soybean cultivars both increased in response to Al toxicity stress but no significant difference between them was observed.
柠檬酸与针铁矿交互作用时会形成针铁矿-柠檬酸的复合胶体，增强对重金属活性离子的吸附，降低重金属毒性。
The adsorption will be enhanced by the goethite-citric acid compound colloid which from the citric acid and goethite interaction.
在之前的研究中，我们发现马兜铃酸是一种强致突变剂，对小鼠淋巴瘤细胞有显著的细胞毒性和致突变作用。
In the previous study, we have found that AA is a potent mutagen which can induce significant cytotoxic and mutagenic effects on mouse lymphoma cells.
结论：适当浓度的亚砷酸可引起神经轴突的损害，且对全身及周围软硬组织无毒性破坏作用。
Conclusion: Suitable arsenic trioxide concentration can damage nerve axons permanently, and can not bring side effect to the structure around the nerve and whole body.
结论：适当浓度的亚砷酸可引起神经轴突的损害，且对全身及周围软硬组织无毒性破坏作用。
Conclusion: Suitable arsenic trioxide concentration can damage nerve axons permanently, and can not bring side effect to the structure around the nerve and whole body.
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