结论：动作电位传导过程中造成的局部负电场能直接始动并参与中枢神经轴突髓鞘化。
CONCLUSION: the local negative electric field that is brought about by action potential can directly initiate and participate in myelination in the CNS.
模型方程分别对应于突触后电位、感受器电位、始段分级电位和轴突上的的脉冲系列，每个方程都具有明确的生理意义。
The equations of the model, corresponding to postsynaptic potentials, receptor potentials, initial segment graded potentials and the impulse train along the axon respectively, have been given.
我们与其他人的研究至此得出了结论：神经胶细胞藉著侦测由激发轴突释放或是从突触渗漏的AT P，而能知道神经元产生了动作电位。
To THIS POINT, work by us and others had led to the conclusion that a glial cell senses neuronal action potentials by detecting ATP that is either released by a firing axon or leaked from the synapse.
结果：雪旺细胞结合纤连蛋白治疗组脊髓诱发电位潜伏期显著缩短，再生轴突明显增多，二者呈负相关。
Results:In the group treated with SCs and FN, the latency of P1 wave was significantly shorten and the number of regrowed axons significant increased, with a significant negative correlation.
结果：雪旺细胞结合纤连蛋白治疗组脊髓诱发电位潜伏期显著缩短，再生轴突明显增多，二者呈负相关。
Results:In the group treated with SCs and FN, the latency of P1 wave was significantly shorten and the number of regrowed axons significant increased, with a significant negative correlation.
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