催产素和血管加压素是情感和社会行为及关系中起重要作用的激素。
Oxytocin and vasopressin are hormones that play key roles in emotional and social behaviors and bonding.
低剂量的传统血管加压素治疗可以降低败血症病人的死亡率…
Would decrease mortality among patients with septic shock who were being treated with conventional vasopressor …
急性出血的主要治疗药物为血管加压素、生长抑素及其类似物。
The main drugs for acute hemorrhage are vasopressin, somatostatin and its analogues.
目的：观察院前心肺复苏中联合应用血管加压素与肾上腺素的疗效。
Objective: To observe the therapeutic effects of vasopressin and adrenalin on cardiopulmonary resuscitation before admitted to hospital.
本研究旨在评价血管加压素v 2受体拮抗剂的急性血流动力学效应。
This study sought to assess the acute hemodynamic effect of vasopressin V2 receptor antagonism.
科学家在研究草原田鼠时，发现了血管加压素在长期恋爱关系中的作用。
It is an important controller of the kidney and its role in long-term relationships was discovered when scientists looked at the prairie vole.
科学家发现这些血管加压素会让这些小老鼠今后的行为和记忆出不同程度的问题。
The researchers were able to show that vasopressin was behind the behavioural and memory problems.
结论：血管加压素、肾上腺素联合纳洛酮在心肺复苏中的应用效果满意，值得临床关注。
Conclusion: Vasopressin, Epinephrine and Naloxone in cardiopulmonary resuscitation have satisfactory result and there are worthy of attention.
目前的一个重要目标是研究催产素和血管加压素调节的神经发育过程，以完全理解它们抗自闭症的能力。
One important goal now is to fully characterize the neurodevelopmental processes modulated by oxytocin and vasopressin in order to fully understand their ability to reverse autistic symptoms.
如果这些结果能被将来的研究证实，那么血管加压素v 2受体拮抗剂可能将成为新的降压策略，尤其是适用黑人种群。
If these results are confirmed in further studies, then vasopressin V2 receptor antagonists might offer a novel antihypertensive strategy, especially in the black population.
如果是因为激素缺少而不是肾小管对这种激素没有反应的话，可注射血管加压素的生物制剂或化学合成制剂来治疗。下视丘的病变也会造成尿崩症。
Injections of synthetic vasopressin are effective if the hormone is lacking but not if the response is absent. disorders of the hypothalamus are one cause of diabetes insipid us.
结论：大鼠尾加压素II是大鼠肺主动脉有效的血管收缩剂，细胞信号转导通路MAPK的激活参与其收缩效应。
CONCLUSION: rat U-II was a potent vasoconstrictor of rat main pulmonary arteries and this response was mediated through MAPK.
结论：大鼠尾加压素II是大鼠肺主动脉有效的血管收缩剂，细胞信号转导通路MAPK的激活参与其收缩效应。
CONCLUSION: rat U-II was a potent vasoconstrictor of rat main pulmonary arteries and this response was mediated through MAPK.
应用推荐