表明三七皂甙对能量代谢障碍引起的神经细胞损伤有保护作用，这一作用可能与其抗脂质过氧化损伤有关。
It is revealed that PNS can prevent the damage or nerve cells due to energy metabolic disturbance, which may relate to its function of fighting against lipid peroxidation.
结论：朊蛋白106- 126肽段对分化PC 12细胞具有毒性作用，能量代谢障碍可能是其毒性作用机制之一。
Conclusion: Prion protein 106-126 peptide has cellular toxicity effect on differentiated PC12 cells, and the energy metabolize disorder may be one of the reasons for the toxic mechanism.
结论：老年2型糖尿病患者血粘度增高和红细胞变形能力降低可以增加代谢物质血流传输障碍，加重胰岛素抵抗。
Conclusion: High blood viscosity and impaired erythrocyte deformability in erythrocyte membrane may increase resistance to insulin in aged type 2 diabetes.
结论：氧中毒后大脑出现能量代谢障碍及细胞内酸碱代谢紊乱。
Conclusion: There are energy failure and intracellular acidosis and alkalosis in cerebral cellular after oxygen poisoning in rats.
结论：苯代谢产物诱导的造血细胞凋亡和坏死可能是苯中毒导致的造血功能障碍的主要机制之一。
Conclusion Metabolites of benzene can induced hematopoietic cell apoptosis and necrosis, and give rise to aplastic anemia.
结论：超微结构变化提示感光细胞能量代谢系统和(或)自噬系统功能障碍。
CONCLUSIONS: These changes suggested that self energizing system and self engulfing system of the photoreceptors were disfunctioned.
提示脑细胞代谢活跃，神经轴突轴浆转运障碍，突触传递受阻。
The results indicated that the metabolism of brain cells was active; the axoplasmic transport of axon was damaged; synaptic transmission function was abnormal.
代谢障碍有关的组织细胞增多症，通常发生在年轻人身上。
Histiocytosis associated with disturbance of cholesterol metabolism, occurs chiefly in young children.
结论：OFR通过增加ADMA导致内皮功能紊乱，卡托普利则能通过减少ADMA减轻OFR诱导的内皮细胞代谢功能障碍。
CONCLUSIONS: OFR induces endothelial dysfunction through increasing ADMA concentration, while captopril relieves endothelial dysfunction induced by ox-LDL through decreasing ADMA concentration.
结论：OFR通过增加ADMA导致内皮功能紊乱，卡托普利则能通过减少ADMA减轻OFR诱导的内皮细胞代谢功能障碍。
CONCLUSIONS: OFR induces endothelial dysfunction through increasing ADMA concentration, while captopril relieves endothelial dysfunction induced by ox-LDL through decreasing ADMA concentration.
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