呼吸机相关性肺损伤研究常用大潮气量和高浓度氧策略的动物模型，使用时间相对较短。
Ventilation-induced lung injury is often studied in animal models by using ventilation strategies with high-tidal volumes and high-oxygen concentration over a relatively short period of time.
结论低潮气量通气治疗ARDS动物模型能够减轻炎症诱发细胞因子的释放，减轻机械通气相关性肺损伤。
Conclusion Low tidal volume ventilation can decrease cytokine release and can alleviate ventilator - induced lung injury (VILI) in animal models of ARDS.
目的通过建立过度通气导致呼吸机相关性肺损伤(VILI)大鼠模型，评估不同潮气量对肺损伤的影响。
Objective to establish an experimental model of ventilator-induced lung injury (VILI) in rats through tracheal tube and evaluate the influence of different ventilatory volumes on VILI in rat lung.
目的探讨允许性高碳酸血症肺保护性通气策略对降低呼吸窘迫综合征新生儿(NRDS)呼吸机相关性肺损伤的价值。
Objective to investigate the advantages of protective ventilation strategies in NRDS and the value of decreasing the incidence of ventilator-associated lung injury (VALI).
结果染毒后小鼠脾、肝、肺、肾组织细胞DNA出现损伤，且损伤程度与染毒剂量具有一定相关性。
Results DNA of spleen, liver, lung and kidney cells was significantly damaged after exposure, and there were some correlation between exposure concentration and damage degree.
目的探讨大鼠急性肺损伤（ALI）过程中，信号转导和转录活化因子-3（STAT3）的活化，及其与细胞因子白介素-6（IL-6）的相关性。
Objective To investigate the activation law of signal transducers and activators of transduction-3(STAT3) and whether cytokine IL-6 can activate STAT3 in acute lung injury (ALI) of rats.
目的探讨大鼠急性肺损伤（ALI）过程中，信号转导和转录活化因子-3（STAT3）的活化，及其与细胞因子白介素-6（IL-6）的相关性。
Objective To investigate the activation law of signal transducers and activators of transduction-3(STAT3) and whether cytokine IL-6 can activate STAT3 in acute lung injury (ALI) of rats.
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