对遗传信息进行编码。基因——长长的脱氧核糖核酸分子中的不连续的片段——把它们的序列转录成单线信使核糖核酸分子，核糖核酸又成为蛋白质的模板。
Genes — discrete segments of long DNA molecules — transcribe their sequences onto single-strand messenger RNA molecules, which then serve as templates for proteins.
基因组中具有调控功能的DNA序列片段可称为转录调控元件。
The DNA segments which have certain regulatory function in genomic sequences are called transcriptional regulatory elements.
对这些片段的DNA序列分析表明，它们均存在各种顺式作用元件，由此推测它们可能是被一些木质素过氧化物酶基因转录调控相关的蛋白质所结合的序列。
Analysis of the sequences showed that there were many cis-regulatory elements in these DNA segments, implying that these sequences may be bound by some transcriptional regulation protein factors.
在多个调控序列上得到的出现频率高的片段很可能是对于这组序列的转录调控有重要作用的序列元件。
Over-represented motifs identified in a group of related regulatory sequences may play an important role in transcription regulation of the genes.
到每个转录本或基因的比对片段的数目在不同条件和重复下变化。
The number of mapped reads to each transcript or gene varies under different conditions and replicates.
用随机引物反转录，通过TC-RT-PCR检测ACLSV和ASGV复合感染的梨样品也均获得了目标片段。
A TC-RT-PCR method basing on reverse transcription with random primer facilitated the detection for those samples mixed infected by ASGV and ACLSV.
前言： 近年来，一些不依赖于转录因子活性的新型双杂交系统相继建立，如分离的泛素系统、 蛋白质片段互补分析、阻遏物重构分析和SOS恢复系统等。
Some new type of two-hybrid systems, such as split-ubiquitin system, protein-fragment complementation assay, repressor reconstitution assay and SOS recruitment system, have been developed recently.
方法设计出针对各片段的特异性引物，用P CR方法从Z 37病毒感染的细胞提取细胞总rna，逆转录扩增、产物克隆t载体，纯化后测序，测定的序列应用DNASTAR软件比较分析。
Methods the total RNA was extracted from Z37 virus infected cells and the RT-PCR products were cloned into t vector, sequenced and analyzed by DNASTAR software.
证实编码逆转录酶第300至311位氨基酸的基因片段具有调控乙肝病毒复制的功能。
It is proved that the gene segment coding the Nos. 300-311 amino acid of the reverse transcriptase possesses the function of controlling the copying of HBV.
证实编码逆转录酶第300至311位氨基酸的基因片段具有调控乙肝病毒复制的功能。
It is proved that the gene segment coding the Nos. 300-311 amino acid of the reverse transcriptase possesses the function of controlling the copying of HBV.
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