炎症性肠病是一个条件，导致炎症和炎性细胞的浸润肠壁。
Inflammatory Bowel Disease is a condition resulting from an inflammatory condition and an infiltration of the gut wall with inflammatory cells.
结论痰液炎性细胞可能成为反映气道炎症类型、鉴别感染后咳嗽与咳嗽变异性哮喘的一项参考指标。
Conclusion Sputum inflammatory cells may be a useful marker reflecting the type of airway inflammation and differing between postinfectious cough and cough variant asthma.
AR与BA的发病机制至今仍不十分清楚，其是涉及多种炎性细胞的相互作用，诸多炎性介质、细胞因子、转录因子等共同参与的慢性炎症反应。
The pathogenesis of AR and BA has not been quite clearly, it may be involved in the interaction of many inflammatory mediators, cytokines and transcriptional factors.
牙周优势菌lps作用于单核巨噬细胞，诱导产生各种细胞因子和炎性介质，参与牙周病的炎症激活和组织创伤。
LPS involves in the inflammatory process and tissue destruction during periodontal diseases through inducing mononuclear macrophages to produce various cell factors and inflammatory mediators.
黄芩苷各治疗组肺组织炎症较免疫抑制对照组明显减轻，肺间质炎性细胞浸润及肺泡腔泡沫样渗出物均明显减少。
Compared with immunosuppressed control group, there were less alveolar interstitial lymphocytes, foamy intra-alveolar exudate and inflammation of lung tissue in rats of drug treatment groups.
结论哮喘病是一种慢性气道炎症性疾病，在不同发病时期气道内的炎性细胞变化不同。
ConclusionAsthma is a kind of diseases characterized by chronic inflammation of airway. The composition of inflammatory cells in the airway was different during the diversity phase of the asthma.
结论：IL5在变应性鼻炎鼻粘膜、鼻息肉组织内浸润的多种炎性细胞中表达，能客观反映免疫或炎症反应的程度，可作为变应性鼻炎诊断评分系统的补充。
Conclusion: IL5 is expressed in allergic rhinitis and nasal polyp tissue. It reflects the degree of immunology and inflammation and can be used as an index to diagnose allergic rhinitis.
结论通心络对大鼠佐剂性关节炎具有显著抑制作用，其作用机制可能与抑制前炎症细胞因子生成、抗氧化等有关。
ConclusionTongxinluo capsule has obvious inhibition effect on AA, and the action mechanism may be related with the antioxidation and inhibition of the production of proinflammatory cytokines.
结论通心络对大鼠佐剂性关节炎具有显著抑制作用，其作用机制可能与抑制前炎症细胞因子生成、抗氧化等有关。
ConclusionTongxinluo capsule has obvious inhibition effect on AA, and the action mechanism may be related with the antioxidation and inhibition of the production of proinflammatory cytokines.
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