探讨缺氧复给氧损伤后脑微血管内皮细胞凋亡的机制。
To explore the mechanism of apoptosis after hypoxia-reperfusion oxygen damage the cerebral microvascular endothelial cells.
氧损伤也称为“氧化损伤”。可以通过抗氧化剂而减少。
Oxygen damage is also called "oxidative damage" and can be reduced by antioxidants.
海马神经元；缺氧复氧损伤；脑神康胶囊；抗氧化；凋亡。
Hippocampal neurons; Anoxiareoxygenation; Naoshenkang capsule; oxygen free radical; Apoptosis.
脑微血管内皮细胞缺氧复给氧损伤后，采用流式细胞仪检测细胞凋亡的程度。
Apoptosis was detected with flow cytometry after hypoxia-reperfusion oxygen damage of the cerebral microvascular endothelial cells.
目的明确异氟醚减少离体鼠肝缺氧-复氧损伤的功能主要发生在哪一阶段，以及和枯否细胞的关系。
Objective To investigate which stage is the most important and the relationship with kupffer cell, during isoflurane decrease anoxia-reoxygenation injury in isolated rat liver.
摘要目的明确异氟醚减少离体鼠肝缺氧-复氧损伤的功能主要发生在哪一阶段，以及和枯否细胞的关系。
Abstract Objective To investigate which stage is the most important and the relationship with kupffer cell, during isoflurane decrease anoxia-reoxygenation injury in isolated rat liver.
细胞形态呈现多样化，如圆形、梭形及椎形。缺氧复氧损伤后心肌细胞伪足缩短或消失，折光性下降，搏动减弱或消失。
ResultsBeing injured by hypoxia and reoxygenation, refracting power of the cells declined, pseudo-podium became shortened or disappeared, cell beat became weak or stopped.
这些结果说明线粒体能量代谢障碍是缺血再灌注损伤的始动环节，而氧自由基是造成损伤的主要因素。
These results showed that the energy metabolism dysfunction was the initial stage, and the damage of oxygen free radicals was the major factor in ischemia-reperfusion injury.
这些问题在男孩子当中更加普遍，尤其是那些在婴儿时期遭受了神经组织损伤，或者身体虚弱到需要通过供氧来维持生命的男孩子们。
These problems were more common among males, those with injured nerve tissue in the brain and those who had been given oxygen supplies for days as a baby.
然而，研究人员发现在30摄氏度这种清除过程并不能清除活性氧或阻止氧化损伤的发生。
However, at 30 degrees centigrade the researchers found that the cleanup processes cannot cope with ROS and oxidative damage occurs.
去年，这个研究小组证明了氢能消除水中活性氧并能使中风对脑的损伤减轻一半。
Last year, the team confirmed that hydrogen removes active oxygen from water and can halve brain damage from strokes.
大动脉置换术前脑损伤与氧饱和度以及外科手术的时间有关，而与房间隔气囊造口术无关。
Preoperative Brain Injury in Transposition of the Great Arteries Is Associated With Oxygenation and Time to Surgery, Not Balloon Atrial Septostomy.
报告携氧液在颅脑损伤低氧血症时应用的实验与临床研究。
The experimental and clinical studies on hypoxia of head injuries treated with oxygen carrying solution were reported.
目的：探讨重度颅脑损伤及脑出血术后患者高压氧治疗和其他多项与治疗相关的影响因素。
AIM: to study multifarious factors on treatment of hyperbaric oxygen (HBO) and other correlative factors in patients with severe craniocerebral injury and cerebral hemorrhage.
结果细胞因子、辐射、氧自由基等可诱导血管内皮细胞凋亡，导致血管损伤；
Results Cytokine, radiation could induce apoptosis of endothelial cells, and caused vascular damage.
目的探讨高氧液预处理对兔心肌缺血再灌注损伤的保护作用。
Objective To study the protective effect of hyperoxic liquid preconditioning in myocardial ischemia-reperfusion injury.
结论高压氧对缺血性脑损伤有显著治疗作用，提高脑红蛋白的表达水平可能是其治疗的机制之一。
Conclusion Hyperbaric oxygen treatment had an obvious effect on ischemic brain injury, and increasing the expression of Neuroglobin might be one of the possible mechanisms of the therapy.
然而，最近的动物实验显示，长时间暴露在氧含量较高(氧过多)的环境可导致类似ARDS的肺长期损伤。
However, recent studies in animals have shown that prolonged exposure to an elevated level of oxygen, or hyperoxia, can cause long-term lung injury that resembles ARDS.
过氧化氢和氧自由墓具有强烈敏化作用，损伤组织。
The hydrogen peroxide and the oxygen free grave have the intense sensitization, the damage organization.
目的：观察术前心理干预对脊髓损伤后行脊髓减压患者围手术期心肌耗氧量的影响。
AIM: to observe the influence of preoperative psychological intervention on the myocardial oxygen consumption in patients after spinal cord injury, who are to be decompressed.
结论：术前心理干预可减少脊髓损伤后行脊髓减压患者围手术期心肌耗氧量。
CONCLUSION: Preoperative psychological intervention can reduce the myocardial oxygen consumption in the perioperative patients after spinal cord injury under decompression of spinal cord.
RA通过降低AECII凋亡、坏死从而对高氧肺损伤具有保护作用。
RA had a protective effect on hyperoxia lung injury by which declined AECII apoptosis and necrosis.
结论淫羊藿苷对氧自由基损伤的大鼠脑线粒体呼吸链具有保护作用。
CONCLUSION Icariin has protective effect on rat cerebral mitochondria injury induced by oxygen free radical.
目的探讨地氟醚预处理对大鼠肝脏缺血再灌注损伤时氧自由基的影响。
Objective To investigate the effect of pretreatment with desflurane on oxygen free radicals in liver ischemic reperfusion injury in rats.
结论过度训练对骨骼肌的损害主要是产生自由基介导的氧应激损伤，最终表现为骨骼肌细胞凋亡和坏死。
Conclusions Overtraining may lead to free radical-mediated injuries to the rats' skeletal muscle, cause apoptosis and necrosis as well.
目的探讨海马及皮层的氧自由基损伤与大鼠学习能力和记忆能力的关系。
Objective To investigate the relationship bet ween the learn and memory on the damaged hippocampus and damaged cortex of rats by oxygen free radicals.
并与碱氧一浴轧蒸工艺的处理效果进行比较，半制品质量指标优于碱氧工艺，强力损伤率明显降低。
The quality index of half finished goods was superior to that of alkali-oxygen one bath pad-steaming process, and the strength damage significantly de-creased.
结论高氧液对神经元缺血损伤具有保护作用。
CONCLUSION Hyperoxia solution can mitigate the cerebral ischemia and reperfusion injury.
目的探讨重型颅脑损伤后脑氧代谢相关参数的变化及临床意义。
Objective To investigate the changes and significance of related parameters on cerebral oxygen metabolism in patients with severe brain injury.
目的探讨重型颅脑损伤后脑氧代谢相关参数的变化及临床意义。
Objective To investigate the changes and significance of related parameters on cerebral oxygen metabolism in patients with severe brain injury.
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