PMB有拮抗内毒素，减轻肝脏损伤的作用。
另设正常动物注射内毒素组和甘氨酸拮抗内毒素组加以对照。
The normal rats injected with LPS or injected with LPS combined with glycine (LPS antagonist) were designed as controls.
PAF受体拮抗剂sri63—441对内毒素和PAF所致肺水肿和肺泡灌洗液细胞学变化有部分改善作用。
PAF receptor antagonist SRI63-441 showed partial protective effects on endotoxin and PAF induced lung edema and BALF cytological changes.
以往的工作显示：内毒素能显著提高SOCS在大鼠肝脏中的表达，这又同内毒素引起的生长激素的拮抗密切相关。
We have previously shown that endotoxin markedly stimulates SOCS gene expression in rat liver, that correlates with observed resistance to growth hormone-signaling during endotoxemia.
分析了多粘菌素B九肽、类脂A结构拮抗剂、抗脂多糖抗体及免疫治疗、抗内毒素多肽以及中药对内毒素的作用。
Comparison on the effect of polymyxin B nonapeptide, lipid A structure antagonist, anti-LPS antibody and immune therapy, anti-endotoxin polypeptide and function of Chinese herb medicines on endotoxin.
结论：两种组胺受体拮抗剂均可缓解内毒素诱导的雏鸡肺损伤。
Conclusion: Histamine receptor antagonists can reduce the lung injury induced by endotoxin.
结论：两种组胺受体拮抗剂均可缓解内毒素诱导的雏鸡肺损伤。
Conclusion: Histamine receptor antagonists can reduce the lung injury induced by endotoxin.
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