目的观察在三维胶原体外培养条件下恶性胶质瘤细胞体外诱导内皮细胞血管生成的作用。
Objective To investigate the effects of malignant glioma cells on the induction of endothelial cells to form tubule-like structure in a three dimensional culture system with collagen in vitro.
研究人员发现，在实验室关闭RAD51分子，杀死恶性胶质瘤细胞，提高了放射治疗的效果。
Researchers discovered that switching off the RAD51 molecule increased the effectiveness of radiotherapy to kill glioblastoma cells in the lab.
研究人员已经建议一些特殊种类的恶性胶质瘤细胞可以繁殖，复制出相同的拷贝从而更加的耐治疗。
Researchers had suggested that some specialised groups of glioblastoma cells can reproduce to make identical copies that are more resistant to treatment.
埃伯哈特和他的同事们研究了恶性胶质瘤的细胞系以发现γ-内分泌酶抑制剂的影响，这种药物可阻断促使肿瘤增长的Notch受体途径。
Eberhart and colleagues studied glioblastoma cell lines to investigate the effects of a gamma-secretase inhibitor, a medication that blocks the Notch receptor, on tumor growth.
鼻腔神经胶质瘤(又被称为嗅母细胞瘤)是一种不常见的恶性肿瘤，被认为起源于嗅上皮的感觉神经感受体细胞。
Esthesioneuroblastoma (also known as olfactory neuroblastoma) is an uncommon malignant neoplasm of the nasal vault believed to arise from neurosensory receptor cells in the olfactory epithelium.
目的：检测血管内皮生长因子(VEGF)和增殖细胞核抗原(pcna)在胶质瘤中表达，并探讨其与肿瘤恶性程度的关系。
Objective: To examine the expressions of VEGF and PCNA in gliomas, and investigate the possible relationship between their expressions and the degree of tumor malignancy.
目的研究p 16基因改变与脑胶质瘤恶性程度分级及肿瘤细胞增殖活性之间的关系。
Objective To study the relationship between P16 gene modification and the malignant grades of brain glioma and the proliferation activity of tumor cells.
目的：研究LRRC4基因在恶性脑胶质瘤细胞系中的表达情况。
Objective To examine the expression absence of LRRC4 gene in glioblastoma cell lines.
目的研究P 16基因改变与脑胶质瘤恶性程度分级及肿瘤细胞增殖活性之间的关系。
Objective To study the relationship between P16 gene modification and the malignant grades of brain glioma and the proliferative activity of tumor cells.
目的探讨人脑胶质瘤中血管内皮细胞生长因子(VEGF)的表达与微血管数量(MVQ)和肿瘤良恶性程度的关系。
Objective to investigate the relationship between the expression of vascular endothelial growth factor (VEGF), the microvessel quantity (MVQ) and the grade in human brain gliomas.
目的利用已经建立的胶质瘤细胞诱导分化相关基因表达谱，筛选胶质瘤恶性进展相关基因。
Objective To screen out genes associated with malignant progression of glioma, based on the established glioma cell differentiation-inducing gene expression profiles.
该受体在介导人恶性胶质瘤 细胞的迁移、存活、生长及血管发生中起重要作用。
The receptor has important function on mediating migration, survival, growth and vasculogenesis of the human malignant glioma cells.
干细胞具有向恶性胶质瘤趋向性迁移的特性，并可能成为恶性胶质瘤基因治疗的理想载体。
Various studies have demonstrated the tremendous tropism of stem cells for malignant gliomas, making these cells a potential vehicle for delivery of therapeutic genes to disseminated glioma cells.
这些中许多将是高恶性程度胶质瘤，高度侵袭性的癌症形式，将癌细胞扩散到整个大脑和脊髓。
Many of these will be high-grade gliomas, a highly invasive form of cancer that sends cancerous cells throughout the brain and spinal cord.
病变在DWI/ADC上确实显示弥散增加，这不同于大细胞肿瘤入淋巴瘤和高度恶性的胶质瘤。
The lesion did demonstrate increased diffusivity on DWI/ADC maps, which would argue against a highly cellular tumor like lymphoma and a high grade glioma.
脑胶质瘤是由神经外胚叶衍化而来的胶质细胞即星形胶质细胞、少枝胶质细胞和室管膜胶质细胞等发生的肿瘤，是颅内最常见的恶性肿瘤。
Glioma is the most common type of primary intracranial tumors in human. It developed from astrocytoma, oligodendroglioma, ependymoma, which were induced by neural ectoblast.
它们组成了几乎百分之八十的癌细胞生长，就像肯尼迪议员的恶性胶质瘤。
What they make up almost eighty percent of cancer growths, like the malignant glioma thats Senator Kennedy has.
最常见的类型的脑癌即恶性胶质瘤以出现类似癌干细胞的脑细胞为特征，这些细胞不是替代损伤细胞，而是形成肿瘤。
The most common type of brain cancer-glioblastoma-is marked by the presence of these stem-cell-like brain cells, which, instead of triggering the replacement of damaged cells, form cancer tissue.
最常见的类型的脑癌即恶性胶质瘤以出现类似癌干细胞的脑细胞为特征，这些细胞不是替代损伤细胞，而是形成肿瘤。
The most common type of brain cancer-glioblastoma-is marked by the presence of these stem-cell-like brain cells, which, instead of triggering the replacement of damaged cells, form cancer tissue.
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