目的探讨二硫化碳染毒对小鼠心肌细胞凋亡的影响。
Objective to study the effect of carbon disulfide infection of mice myocardial cell apoptosis.
目的研究病毒性心肌炎(V MC)小鼠心肌细胞凋亡及其意义。
Objective to investigate cardiomyocyte apoptosis in viral myocarditis (VMC) mice and its relationship with VMC.
目的研究急性分离小鼠心肌细胞肿胀激活氯离子通道的单通道特征。
AIM: To characterize the single channel properties of swelling-activated chloride channel in freshly isolated mouse cardiac myocytes.
目的探讨急、慢性病毒性心肌炎和感染性扩张型心肌病小鼠心肌细胞凋亡的动态变化。
Purpose to investigate dynamic alteration of apoptosis in myocardium of mice with acute, chronic myocarditis and dilated cardiomyopathy.
研究表明在小鼠研究中，该病毒感染心肌细胞所使用的受体是正常心脏跳动所必须的。
The researchers demonstrated that the receptor which the virus uses to infect heart cells is normally necessary for regular heart beat in mice.
目的：研究小鼠胚胎干细胞分化心肌细胞钠通道、钙通道电生理特性。
AIM: To study the electrophysiological characteristics of ion channels of stem cell derived cardiomyocytes (SCDC) of mouse.
在小鼠身上的研究表明，运动开启遗传计划导致心脏生长，心肌细胞分离。
The studies in mice suggest that exercise turns on a genetic program that leads the heart to grow as heart muscle cells divide.
结论实验组小鼠在接种CV B3后可引起VMC, VMC中存在异常的心肌细胞凋亡现象。
Conclusions VMC can be caused in mice after CVB3 inoculation, abnormal cardiomyocyte apoptosis can be found in VMC.
结论小鼠及大鼠一次性急性力竭运动可致心肌细胞水肿与线粒体损伤。
Conclusion Acute exhaustive exercise can induce myocardial edema and mitochondrial damage in mice and rats.
方法：连接蛋白43和在实验小鼠病毒性心肌炎心肌细胞结蛋白表达免疫测定。
METHODS: The expression of connexin 43 and desmin in cardiac muscle cells of mice with experimental viral myocarditis was determined by immunohistochemistry.
结论：本法无需添加化学诱导剂，为一种简便、经济的诱导体外培养小鼠es细胞分化为心肌细胞的方法。
CONCLUSION: a simple and economical method was established to induce mouse es cells cultured in vitro to differentiate into cardiomyocytes without using any chemical reagents.
结论：本法无需添加化学诱导剂，为一种简便、经济的诱导体外培养小鼠es细胞分化为心肌细胞的方法。
CONCLUSION: a simple and economical method was established to induce mouse es cells cultured in vitro to differentiate into cardiomyocytes without using any chemical reagents.
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