虽然身高是一种明显的遗传特征，但是个体的实际身高则取决于基因和环境的相互作用。
While tallness is evidently a hereditary characteristic, any individual's actual height depends on the interaction between their genes and the environment.
基因和环境以许多复杂的方式相互作用，塑造个体并指导他们的行为，包括他们的好奇。
Genes and the environment interact in many complex ways to shape individuals and guide their behavior, including their curiosity.
这和其他的资料共同声称决定一个个体是否会患上抑郁的是基因-环境相互作用。
This and other data was used to argue that what determines whether an individual will get depression is a gene-environment interaction.
像其他精神疾病一样，酒精使用障碍(AUDs)是长期多种基因弱点和环境因素相互作用形成的。
Like other mental illnesses, alcohol use disorders (AUDs) develop through interactions of multiple genetic vulnerabilities and environmental factors over a long period.
高血压是脑卒中、心力衰竭、肾功能衰竭的最主要发病因素，是多基因遗传和多个环境因素相互作用的结果。
Hypertension is the main cause for cerebral apoplexy, heart failure and renal failure, and is the consequence of the interaction of polygenic inheritance and environmental factors.
系统生物学通过整合各种“组学”来研究基因，分子和环境层面的各种相互作用对代谢类疾病的影响。
Systems-based approaches tackle the complex interplay of genetic, molecular, and environmental factors contributing to metabolic diseases by integrating a range of "omics" data.
大多数疾病—包括糠尿病和一号杀手，心脏病—是由复杂的多基因和环境因素相互作用引起的，如日常饮食，锻炼和吸烟。
Most diseases - including diabetes and the No. 1 killer, heart disease - are caused by complex interactions of multiple genes and environmental factors, such as diet, exercise and smoking.
项目的目的之一是依靠观察生活方式和环境如何同基因一起随着时间相互作用，从而找到一系列儿童疾病的遗传致病因素。 但是一个关键因素是研究者药经常和受试者沟通。
One of the project's goals is to pinpoint the genetic causes of a range of childhood diseases by seeing how lifestyle and environment interact with genetic risk over time.
统计基因技术被用来确定癌风险中的遗传形式，基因定位和评估基因-环境的相互作用（70年代到90年代）。
Statistical genetic techniques developed to define modes of inheritance, localize genes, and evaluate gene-environment interactions in cancer risk (1970s through 1990s).
统计基因技术被用来确定癌风险中的遗传形式，基因定位和评估基因-环境的相互作用（70年代到90年代）。
Statistical genetic techniques developed to define modes of inheritance, localize genes, and evaluate gene-environment interactions in cancer risk (1970s through 1990s).
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