结论灯盏花素可通过抑制神经细胞凋亡而减轻缺血再灌注引起的脑损伤。
CONCLUSION Breviscapine showed an improvement effect against brain impairment induced by ischemia-reperfusion in mice, and the inhibition of apoptosis in brain may be involved in.
利多卡因可减轻缺血再灌注和炎症反应所致的细胞损伤，其机制至今不明。
Lidocaine attenuates cell injury induced by ischemic-reperfusion and inflammation, although the protective mechanisms are not understood.
结论：缺血后处理可减轻缺血再灌注损伤、其机制可能与减少心肌细胞凋亡有关。
CONCLUSION: ischemic postconditioning can relieve the ischemic reperfusion injury, the mechanism of which may be related with the decreased myocardial apoptosis.
因此，我们通过鼠来研究是否吸入氢气减轻缺血再灌注损伤来起到心脏保护作用。
Therefore, we investigate whether H2 gas confers cardioprotection against ischemia–reperfusion injury in rats.
因此，在冠脉血运重建时，氢气吸入将成为一种减轻缺血再灌注损伤的非常有希望的措施。
Inhalation of H2 gas at incombustible levels during ischemia and reperfusion reduces infarct size without altering hemodynamic parameters, thereby preventing deleterious left ventricular remodeling.
结论：大蒜素可直接清除氧自由基，可减轻缺血再灌注损伤，为其治疗缺血性心脑血管疾病提供了实验室依据。
Conclusion: Garlicin can directly scavenge oxygen free radicals and improve ischemia reperfusion injury in the cardiovascular and cerebrovascular diseases.
心肌缺血再灌注损伤时E-选择素表达明显增加，降低E选-择素的表达或阻断其作用可减轻缺血再灌注损伤。
The expression of E-selectin can be increased significantly during ischemia-reperfusion injury. A protective effect can be obtained by blocking the expression of ligands of E-selectin.
结论：缺血再灌注视网膜损伤与视网膜组织tpa含量升高有关，亚低温可通过降低视网膜TPA含量而减轻缺血再灌注视网膜损伤。
CONCLUSION: the retina injury induced by IR is related with the increasing content of TPA, mild hypothermia can protect the retina induced by IR injury by decreasing the content of TPA.
结论血栓通能减轻脑组织的缺血再灌注损伤，改善神经功能缺失，其作用机制可能与增加HSP70表达有关。
Conclusion Xueshuantong can protect brain tissue from cerebral ischemia reperfusion injury and improve the nerve function. The mechanism may be related with the increasing of HSP70 expression.
结论：丹皮酚可能具有抑制大鼠脑缺血再灌注后ICAM -1蛋白表达的作用，从而减轻了神经元损伤。
CONCLUSION: Paeonol may inhibit ICAM 1 protein expression after focal cerebral ischemia reperfusion in rats so as to relieve neuron damage.
结论亚低温能减轻大鼠脑缺血再灌注损伤，降低脑组织HSP70及GFAP蛋白的表达。
Conclusion Mild hypothermia therapy may inhibit the expression of HSP70 and GFAP after cerebral ischemia and reperfusion injury in rats.
结论：硬膜外阻滞复合异氟烷吸入，可减轻手术应激反应，对肝脏缺血再灌注损伤有一定的保护作用。
Conclusions: Epidural anesthesia combined with isoflurane can reduce the stress response and produce protective effects on the hepatic ischemia-reperfusion injury to some extent.
目的：研究脑泰方提取物预处理减轻沙土鼠脑缺血再灌注损伤的作用及作用机理。
Objective To study the effects and mechanism of Naotaifang extract preconditioning on the cerebral ischemia-reperfusion injury in gerbil.
此外它还具有广泛的药理学作用，例如防止氧化应激损伤，减轻炎症反应，降低血压，抗缺血再灌注损伤等。
It has broad pharmacological functions such as to prevent oxygenated stress damage, to diminish inflammation, to drop high blood pressure and to resist ischemia reperfusion injury.
目的寻找有效的减轻供肝缺血再灌注损伤的途径。
Objective to explore an effective way to alleviate ischemia reperfusion injury of the donor liver in liver transplantation.
结论：诱导HO - 1可通过抑制细胞凋亡而减轻移植肠缺血再灌注损伤。
Conclusion: HO-1 can alleviate the ischemia-reperfusion injury of transplanted small bowel by reducing apoptosis.
结论抑制枯否细胞活化可减轻肝脏缺血再灌注损伤，枯否细胞在肝脏缺血再灌注损伤中的作用很重要。
Conclusion Inhibiting activation of Kupffer cells may attenuate hepatic ischemia reperfusion injury, and Kupffer cells may play an important role in hepatic ischemia reperfusion injury.
结论：心肌肽素通过抑制心肌细胞凋亡，减轻心肌细胞结构损伤发挥对大鼠心肌缺血再灌注损伤的保护作用。
CONCLUSION: Cardiomyopeptidin plays a role in protecting myocardial ischemia-reperfusion injury in rats by inhibiting myocardial apoptosis and alleviating myocardial structural lesions.
在肝脏移植物获取前，提高供肝内热休克蛋白含量，将明显减轻移植肝脏缺血再灌注损伤。
And it can protect the liver ischemia-reperfusion injury obviously, before the graft harvest, increasing the expression of HSP of liver will lighten the liver ischemia-reperfusion injury.
结论PD 142893能减轻大鼠肺移植早期缺血再灌注损伤，机制与减轻肺间质水肿及炎症反应有关。
Conclusion Treatment of lung allografts with PD142893 can ameliorate ischemia - reperfusion injury by alleviating lung interstitial edema and inhibiting inflammatory reaction.
结论黄体酮可以降低缺血再灌注大鼠脑组织AQP4的表达，从而降低血脑屏障的通透性，减轻脑水肿。
Conclusion Progesterone could alleviate cerebral edema via reducing permeability of blood brain barrier by the inhibition on protein expression of AQP4.
一定浓度吡那地尔预处理能减轻家兔心室肌缺血再灌注所致触发性心律失常，其机制可能与减轻EAD有关；
Conclusion: (1) The ischemia or reperfusion arrhythmias could be suppressed by pinacidil preconditioning and mechanism may relate to eliminate early afterdepolarization (EAD).
结果：三七总皂苷可有效降低脑缺血再灌注损伤大鼠的脑梗死体积，明显减轻脑缺血区血脑屏障破坏的程度。
Result: PNS could reduce infarct volume of cerebral ischemia-reperfusion injury in rats, reduce the extent of the damage in blood-brain barrier significantly.
结论：尼古丁减轻脑缺血再灌注大鼠神经损伤。
Conclusion: Nicotine reduces neural deficit induced by ischemia reperfusion in rats.
结论：缺血后处理可以减轻供体肺的缺血再灌注损伤，改善供体肺功能。
CONCLUSION: ischemic postconditioning effectively improves the respiratory functions by attenuating lung ischemic reperfusion injury.
结论：康络素通过抑制谷氨酸释放可减轻脑缺血再灌注期间脑组织损伤。
Conclusion: Cronassial can inhibit Glu and protect brain tissue during cerebral ischemia reperfusion.
结论：康络素通过抑制谷氨酸释放可减轻脑缺血再灌注期间脑组织损伤。
Conclusion: Cronassial can inhibit Glu and protect brain tissue during cerebral ischemia reperfusion.
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