再灌注期间，上皮细胞功能障碍，内源酶激活，白细胞聚集并活化，都会导致氧自由基的产生。
During the reperfusion period, endothelial dysfunction, activation of endogenous enzymes, leucocyte recruitment and activation all lead to the generation of oxygen -derived free radical.
线粒体呼吸链电子漏是运动性内源活性氧生成的重要机制之一。
The mitochondrial electron leak is an important source of endogenous ROS in exercise.
活性氧代谢还与植物内源激素、丙二醛含量和电解质渗漏率及果实品质变化有密切关系。
The active oxygen metabolism is significantly related to the plant hormones, content of malondialdehyde (MDA), cell electrolyte leakage and quality of fruits.
外源ABA处理增加内源ABA含量，加快内源IAA的降解，促使脂氧合酶活性峰值提前出现，加速果实软化；
Exogenous ABA treatment promoted the increase in endogenous ABA content, quickened the degradation of endogenous IAA, increased lipoxygenase activity and accelerated fruit ripening and softening.
目的探讨慢性肺心病肺动脉高压患者中血红素氧合酶(HO) - 1和内源性一氧化碳(CO)的变化。
Objective To study the changing of heme-oxygenase-1 (HO-1) and endogenous carbon-monoxide (HbCO) in patients with chronic pulmonary hypertension.
结论：心肌缺血预处理与电针内关通过提高内源性氧自由基清除系统酶的活性，抑制缺血再灌注心肌细胞膜脂质过氧化反应，对缺血再灌注心肌具有明显的保护作用。
Coneclusion:EA at Neiguan point and IC can protect Z/R myocardium evidently by raising the activity of inhabiting cell membrane lipid peroxidation of myocardial suffered from ischemic reperfusion.
结论：心肌缺血预处理与电针内关通过提高内源性氧自由基清除系统酶的活性，抑制缺血再灌注心肌细胞膜脂质过氧化反应，对缺血再灌注心肌具有明显的保护作用。
Coneclusion:EA at Neiguan point and IC can protect Z/R myocardium evidently by raising the activity of inhabiting cell membrane lipid peroxidation of myocardial suffered from ischemic reperfusion.
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