• The blocking of dengue virus replication was observed by immunofluorescence.

    然后通过免疫荧光法,观察对登革病毒复制的阻断作用。

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  • Current drugs for HIV work by blocking one of the steps toward virus replication.

    目前的HIV治疗药物都是阻断病毒复制的一个步骤而已。

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  • However, when these cells are activated, the hidden virus replication will restart.

    然而,当这些细胞被激活时,隐藏的病毒便重新开始复制。

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  • AIM: to demonstrate the effect of HCV NS5A expression on vesicular stomatitis virus replication.

    目的:丙型肝炎病毒NS5A的表达对水泡性口炎病毒复制的。

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  • In addition, animal experiments show that NKT cells could induce hepatocyte injury and inhibit hepatitis virus replication.

    另外,动物实验显示NKT细胞可致肝组织损伤并能抑制肝炎病毒的复制。

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  • Conveniently, the microRNA that targets t antigen is made late in the infectious cycle, just when t antigen is no longer essential for virus replication.

    以t抗原为攻击目标的小分子rna在感染周期的最后环节才会产生,而这时t抗原已经不再是病毒繁殖的必须条件。

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  • The formation of CIC may not benefit to the control of virus replication and the induction of anti-virus immunity; CIC has a role in the pathogenesis after SIV infection.

    CIC的形成可能无益于控制病毒复制和引导抗病毒免疫,并可能参与了SIV感染后的发病过程。

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  • Recent studies have found that by HIV-infected macrophages and dendritic cells may also play a role in drug therapy may not completely prevent virus replication in these cells.

    最近研究人员发现,受HIV感染的巨噬细胞和树突状细胞可能也发挥了作用,药物治疗也许并不能完全阻止病毒在这些细胞中复制。

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  • Virus replication can persist in such patients for prolonged periods of time despite antiviral treatment, creating an environment in which drug-resistant viruses can readily be selected.

    病毒复制可延长这类患者的患病时间,尽管他们在接受抗病毒治疗,从而产生一种耐药性病毒很容易适应的环境。

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  • Many studies show that 4-oxo-dihydroquinolines have good oral bioavailability and plasma clearance, and they suppress both the DNA polymerase of HSV and virus replication in the cells.

    多项研究表明,4 -氧代二氢喹啉类有良好的口服利用率及中等程度的血浆清除率,不仅对核苷酸聚合酶产生抑制,还可以抑制细胞内的病毒复制。

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  • While HAART is really effective in keeping down active replication, minocycline is another arm of defense against the virus.

    虽然HAART降低活跃的复制很有效,二甲胺四环素是对抗病毒的另一重要手段。

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  • The virus infects the fatty tissue and increases replication, differentiation, and accumulation of fat cells.

    该病毒会感染脂肪组织,然后复制、分化、积累脂肪细胞。

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  • This strategy, known as pre-exposure prophylaxis, or PrEP, is based on the concept that blocking HIV's replication immediately after exposure to the virus may prevent infection.

    这一策略被称为“暴露前预防”(PrEP),是建立在这样的概念基础上:在接触病毒之后立即阻断HIV的自我复制可以预防感染。

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  • RESULTS: Baogan pills could improve liver function and inhibit replication of hepatitis virus, especially could improve the early parameters of hepatocirrhosis.

    结果:保肝丸有改善肝功能和抑制病毒复制的作用,尤其对肝硬化的早期指标有改善作用。

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  • This has allowed identification of important details in its replication cycle and how the virus interacts with its host.

    这使得确定的重要细节在其复制周期和病毒如何与宿主相互作用。

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  • Instead, the virus required cell activation for replication and mediated cell fusion of t lymphocytes.

    相反,该病毒需要细胞激活介导的复制和细胞融合的T淋巴细胞。

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  • This level of replication sufficient to infection near the T-lymphocytes, so that a period of dormancy memory t cells in virus hidden library has been expanded.

    这样的复制水平足以感染临近的T淋巴细胞,使休眠期记忆性T细胞中的病毒潜藏库不断得到扩充。

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  • EB inhibited influenza virus-induced cell death and replication in cell culture, the authors report, without inducing significant cytotoxicity in the cultured cells.

    作者报道说,EB在细胞培养中抑制流感病毒诱导的细胞凋亡和复制,而且没有显著的细胞毒性。

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  • Eb treatment inhibited viral replication by binding to influenza virus hemagglutinin, thereby preventing attachment of influenza virus to cells, the report indicates.

    报道还指出,EB治疗是通过与流感病毒血凝素结合,抑制病毒复制,从而阻止了流感病毒与细胞的黏附。

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  • AIM: To investigate the effect of mycophenolate acid (MPA) on hepatitis B virus (HBV) replication in vitro.

    目的:探讨霉酚酸(MPA)在体外对乙型肝炎病毒(HBV)复制的影响。

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  • During RNA replication - the phase during which the virus "reproduces" - all three of the subunits of the protein assemble themselves in a particular configuration.

    RNA复制期间即病毒处于增值期蛋白质的所有亚单位组装成一个特殊的结构。

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  • Nature magazine published online a research paper on Wednesday, claiming that virus-specific cells, CD8 + t, are able to control viral replication in both animal models and HIV infections.

    星期三,《自然》杂志发表了一篇研究成果,指出CD 8 +T细胞亚类能抑制动物模型和艾滋病感染的病毒复制。

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  • Using this system, his group has demonstrated sustained replication and production of virus particles for at least 2 months, with these virus particles able to infect new cells.

    他们已经证实,采用这些培养系统可至少维持病毒颗粒复制繁殖2个月,并且这些病毒又能重新感染新的细胞。

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  • With immunosuppression, the dormant JC virus in these cells may be reactivated and start active replication, causing cell death and tissue destruction.

    在免疫抑制时,这些细胞中的大部分病毒可能重新活化,开始活跃复制,导致细胞死亡和组织破坏。

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  • When sera from patients infected with different HCV strains were added to the hepatocyte culture system, viral replication occurred and new virus particles were produced.

    发现当HCV携带者的血清加入该培养系统后,仍有大量病毒复制繁殖。

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  • When sera from patients infected with different HCV strains were added to the hepatocyte culture system, viral replication occurred and new virus particles were produced.

    发现当HCV携带者的血清加入该培养系统后,仍有大量病毒复制繁殖。

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