• Remicade belongs to a class of drugs called tumor necrosis factor (TNF) blockers.

    Remicade属于被称为肿瘤坏死因子(TNF)阻断剂类药物。

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  • To assist in the diagnosis, those being treated with TNF blockers should tell their health care professionals where they live and what areas they have recently visited.

    为了有助于诊断,使用TNF阻滞剂的患者应该告知他们的健康保健专家自己住在什么地方和最近去过那些地点。

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  • TNF and insulin levels were highly correlated.

    肿瘤坏死因子与胰岛素水平有强相关性。

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  • Cytokines such as TNF may prevent transcription of the thrombomodulin gene.

    TNF等细胞因子可阻止凝血调节因子基因的转录。

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  • SIR is relative to inducing and expression of some of cytokines such as ET and TNF .

    全身炎症反应与一些细胞因子如内皮素、肿瘤坏死因子等的诱导表达有关。

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  • Conclusion: Reactive oxygen intermediates was a mediator of apoptosis induced by TNF.

    结论:活性氧中间产物是TNF诱导细胞凋亡的中介体之一。

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  • TNF might play an important role in early stage of inflammatory demylinat ing diseases.

    TNF可能在炎性脱鞘病发病初期起作用。

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  • Objective To study the changes of serum TNF and SA in patients with rheumatic arthritis.

    目的探讨风湿性关节炎患者血清中TNF和SA含量的变化。

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  • Aim to study the effects of TNF receptor blocking peptide on adjuvant arthritis in rats.

    目的观察肿瘤坏死因子(TNF)受体封闭肽对大鼠佐剂性关节炎的影响。

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  • TNF induced in the cell culture was demonstrated by monoclone antibody neutralizing test.

    诱生出的TNF通过单克隆抗体中和试验得到确证。

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  • Conclusion: TNF may be involved in the pathological processes of both CHD and hypertension.

    结论:TNF可能参与冠心病、高血压的病理过程。

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  • Plasma endotoxin, serum TNF, IL-6 and IL-8 were detected at different time phase after burn.

    并在烧伤后不同时相点测定了血浆内毒素、血清TNF、血浆IL-6和血浆IL-8的含量。

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  • TNF related apoptosis inducing ligand (TRAIL) can induce specific apoptosis of cancer cells.

    肿瘤坏死因子相关的凋亡诱导配体(TRAIL)能选择性诱导肿瘤细胞凋亡。

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  • Conclusion: TNF gene may be correlated with the susceptibility of thymoma and thymoma with MG.

    结论TNF基因多态性与胸腺瘤发病及胸腺瘤发生重症肌无力相关。

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  • The changes of CEA and TNF in the pleural effusion were examined before and after the treatment.

    并检测治疗前后胸水中癌胚抗原和肿瘤坏死因子的改变。

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  • Objective To explore the relationship between infection of hepatic virus and free radical and TNF.

    目的探讨肝炎病毒感染与自由基及肿瘤坏死因子的关系。

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  • Patients who develop a fungal infection may be advised to stop the TNF blocker until they recover.

    受到真菌感染的患者可能会被建议在真菌疾病康复前中止使用TNF阻滞剂。

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  • Objective To study the effect of ischemic preconditioning on the apoptosis and the TNF plasma level.

    目的探讨缺血预处理对心肌细胞凋亡及血浆TNF水平的影响。

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  • Lower levels of TNF and higher levels of SOD are apparently beneficial to wound healing after trauma.

    内源性低浓度tnf及抗氧化能力的提高,对创面修复是有益的。

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  • Tumor necrosis factor (TNF) is a potent cytokine that elicits a broad spectrum of biological responses.

    肿瘤坏死因子tnf是一种多功能的细胞因子,具有广泛的生物学活性。

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  • Patients taking TNF blockers should be aware that they are more susceptible to serious fungal infections.

    使用TNF阻滞剂的患者应该注意到他们更易受到严重的真菌感染。

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  • Other risk factors were age, the first year of anti-TNF mAb treatment, and being born in an endemic area.

    其他的危险因素有年龄、抗TNF单抗治疗第一年及出生在结核流行区。

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  • A proliferation-induced ligand(APRIL)is a recently found member of tumor necrosis factor (TNF)superfamily.

    增殖诱导配体(APRIL)是近年来发现的肿瘤坏死因子(TNF)超家族成员。

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  • Objective To study the relationship between nitric oxide(NO), tumor necrosis factor(TNF) and acute pancreatitis.

    目的探讨一氧化氮(NO)和肿瘤坏死因子(TNF)在急性胰腺炎中的作用。

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  • Objective: to find out the relationship between the TNF induced superoxide anion generation and its cytotoxicity.

    目的:了解氧自由基的产生与TNF抗肿瘤活性之间的关系。

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  • Conclusion the plasma TNF level closely correlates with the complicated infectious shock and with the organ failure.

    结论血浆TNF水平与并发感染性休克或器官功能衰竭密切相关。

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  • Results The average TNF content in sera from those patients was significantally higher than that from normal people.

    结果多系统器官功能衰竭组血清TNF含量较正常对照组明显增高,并随病情恶化而升高、病情好转而降低。

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  • Conclusion: Manzhi Kechuanning Capsules can control chronic inflammatory reaction by reducing the contents of TNF and ET.

    结论:慢支咳喘宁胶囊可降低TNF、ET含量,控制慢性炎症反应。

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  • We hypothesized that TNF induces opposing inflammatory and remodeling responses in HF that are TNF-receptor (TNFR) specific.

    我们的研究假设是TNF通过特异的TNF受体(TNFR)诱导相反的炎症和重构反应。

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