But the mechanism of PKB regulation has not been fully elucidated.
目前PK B的相关调控机制还未得到完全阐明。
The nonphosphorylation- level of PKB remained constant in all test.
各组的非磷酸化PKB水平始终保持不变。
We are working on a novel approach to prevent PKB actually binding to the cell membrane.
我们在做的工作就是用一个新途径来抑制蛋白激酶与细胞膜的结合。
Conclusion: Low fat diet and exercise can significantly elevate expression of PKB and ameliorate the state of insulin resistance.
结论：低脂饮食和有氧运动能改善胰岛素抵抗，可能与增加脂肪组织中PK B蛋白表达有关。
The mechanism may be involved in their affecting the phosphorylation and expression of insulin signaling proteins as well as PKB activity.
其作用机制与影响胰岛素信号通道各蛋白的磷酸化、表达及PKB活性等因素有关。
Objective:To study the effect of low-fat-diet and exercise on expression of protein kinase B(PKB) in adipose tissue of rats fed by high-fat -diet.
目的：观察低脂饮食和有氧运动对高脂大鼠脂肪组织中蛋白激酶B（PKB）表达的影响。
CONCLUSION: Aerobic exercise can improve the insulin resistance, the level of PKB expression in liver cells may be related with insulin resistance.
结论：有氧运动能改善胰岛素抵抗，肝细胞中蛋白激酶B表达水平与胰岛素抵抗可能有关。
PKB is considered as a novel and excellent target for antitumor chemotherapy and much attention have been paid to drug development based on PKB target.
PKB是一新的、良好的抗肿瘤药物靶点，以PKB为靶点的药物开发已成为当前研究的热点。
The results suggest that the activated activities for platelet PKB was the causes of unusual activated platelet and platelet thrombosis in coronary heart disease.
冠心病患者血小板PKB高活化性与血小板异常活化及血小板血栓形成有关。
CONCLUSION: EGCG could inhibit lens epithelial cell proliferation by regulating PKB phosphorylation - level, and EGCG has little effect on the total kinase protein.
结论：EGCG可能通过调控PK B的磷酸化水平而抑制晶状体上皮细胞的增殖，并不影响总蛋白含量。
Recent research has shown that certain types of genetic damage, common to many cancer cells, lead to the movement of PKB from the interior of the cell to its surface membrane.
近来的研究显示，某些类型遗传损伤是很多肿瘤细胞的共同原因，导致蛋白激酶从细胞内部运动到细胞膜的表面。
Recent research has shown that certain types of genetic damage, common to many cancer cells, lead to the movement of PKB from the interior of the cell to its surface membrane.
近来的研究显示，某些类型遗传损伤是很多肿瘤细胞的共同原因，导致蛋白激酶从细胞内部运动到细胞膜的表面。
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