• Methods: The different concentrations of anti VEGF antibody was used to act on SKOV3 in vitro.

    方法采用体外实验的方法将不同浓度血管内皮生长因子抗体作用卵巢癌细胞系skov3

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  • Methods SKOV3 cells were incubated with normoxia, hypoxia, and hypoxia plus rapamycin, respectively.

    方法SKOV3细胞分为常组、低低氧雷帕霉素组

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  • Objective To investigate the apoptosis of cultured human ovarian carcinoma cell SKOV3 induced by lobaplatin in vitro.

    目的探讨洛泊对体外培养浆液性卵巢癌细胞SKOV3凋亡诱导作用。

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  • So MDR1 expression in SKOV3/mdr1 cell line was partially inhibited after MDR1-AS treatment in the mRNA and protein level.

    所以导入MDR1基因硫代反义寡聚脱氧核苷酸可以部分抑制SKOV3/mdr1细胞MDR1基因的表达

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  • To observe the effects of Vitamin K3 (VK3) on cell growth and apoptosis in human ovarian cancer cell line SKOV3 and to explore its mechanism.

    观察维生素k3 (VK3)卵巢癌细胞株skov3增殖凋亡影响探讨作用机制。

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  • This article evaluates the effect of heparanase gene on the proliferation, adhesion and invasion of human ovarian cancer cell line SKOV3 cells.

    本文探讨乙酰肝素酶基因转染卵巢癌SKOV3细胞后对细胞生长黏附侵袭能力影响

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  • In this study, we aim to evaluate the inhibitory effect of endostatin on SKOV3 cell and to investigate the possible mechanism of the inhibition.

    目的:内皮素是一种特异性血管内皮生长抑制因子,研究目的在于探讨人内皮素对卵巢癌skov3细胞生长抑制作用及其机制

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  • Methods: The high malignant obtained from ascites ovarian carcinoma cells HRA, SKOV3 and low malignant carcinoma cell 3ao were cultured in vitro.

    方法体外培养腹水来源的人卵巢癌细胞株hraSKOV3性实体瘤来源的人卵巢腺癌细胞3ao

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  • Objective: to develop a human ovarian carcinoma SKOV3 model in severe combined immunodeficiency (SCID) mouse and to study its biologic characteristics.

    目的建立卵巢癌细胞株skov3严重联合免疫缺陷小鼠(SCID)动物模型

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  • To investigate the delayed release effect and the availability of the paclitaxel-loaded PLLA-PEG750 ultrafine fibers on ovarian cancer SKOV3 cell line.

    探讨担载紫杉醇纤维卵巢癌细胞SKOV3缓释效应及作用的有效性

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  • The morphology, growth pattern and CA125 secretion of primary culture of transplanted cells remained as same as those of ovarian carcinoma cell line SKOV3.

    移植后肿瘤细胞形态生长分泌CA12 5功能方面均与原细胞株保持一致

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  • CONCLUSION: Transfected CAR gene may enhance the adhesion ability of ovarian cancer SKOV3 cells, therefore, inhibit the malignant metastatic phenotype of SKOV3 cells.

    结论外源性CAR基因导入可以增强卵巢癌细胞SKOV3间粘附性,从而抑制肿瘤细胞的侵袭转移表型

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  • Objective:To observe caesalpinia sappan aqueous extract's growth inhibiting effects and investigated the mechanism of apoptosis-inducing on human ovarian cancer cell line SKOV3.

    目的观察苏木水提物卵巢癌细胞SKOV3生长抑制作用探讨其诱导靶细胞凋亡及作用机制

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  • AIM: to investigate the inhibitory effects of recombinant human Mullerian inhibiting substance on cell proliferation in human ovarian carcinoma cells (OVCAR8 and SKOV3 cell lines).

    目的探讨重组抑制卵巢癌细胞株ovcar8SKOV3细胞生长影响

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  • The pulse generator was verified that it is easy to adjusted to satisfy the biomedical experiments. And nanosecond pulse electric field can effectively induce apoptosis of SKOV3 cells.

    生物医学实验验证该仪器性能稳定、参数调节方便,证实了脉冲电场能够有效诱导人卵巢腺癌SKOV3细胞凋亡。

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  • To observe the effects of angiostatin gene therapy combined with chemotherapy on intraperitoneal tumor and ascites fluid induced by human ovarian epithelial carcinoma cell line SKOV3 in nude mouse.

    研究血管抑素基因联合化疗药物顺铂卵巢癌细胞SKOV3诱导的裸鼠腹腔肿瘤腹水的抑制作用及其相关机理。

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  • To observe the effects of angiostatin gene therapy combined with chemotherapy on intraperitoneal tumor and ascites fluid induced by human ovarian epithelial carcinoma cell line SKOV3 in nude mouse.

    研究血管抑素基因联合化疗药物顺铂卵巢癌细胞SKOV3诱导的裸鼠腹腔肿瘤腹水的抑制作用及其相关机理。

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