OBJECTIVE: To produce experimental OGD models in cultured neurons.
目的：在神经元培养时制作实验性缺氧缺糖模型。
Neurons were assigned randomly into control group, hypoxia group, OGD group, MK-801 group and d-APV group.
将神经细胞随机分为正常对照组、单纯缺氧组、无糖缺氧组、MK- 801组和d - APV组。
Methods An ischemic model was developed in PC12 cell line with treatment of oxygen glucose deprivation (OGD).
方法采用氧葡萄糖剥夺(ogd)方法在PC 12细胞上建立缺血模型。
To investigate the protective effect of isoflurane on the oxygen and glucose deprivation(OGD) in cultured neuronal cell.
观察异氟醚对氧糖剥夺离体神经元损伤的保护作用。
DAPI staining was used to investigate the role of HPK1 antisense oligodeoxynucleotides in hippocampus neuron death induced by OGD.
采用DAPI染色技术观察hpk1反义寡核苷酸对OGD诱导的海马神经元凋亡的作用。
Objective To understand the protective effect and possible mechanism of Lig on cultured rat astrocytes in oxygen-glucose deprivation (OGD) in vitro.
目的探讨川芎嗪对离体大鼠星形胶质细胞在糖氧剥离损伤中的作用及可能机制。
Conclusion Baicalin possesses protective effect on OGD injury in rat brain slices, which is related to its inhibitory effect on excitatory amino acids toxicity.
结论黄芩苷对大鼠脑片缺氧缺糖性损伤具有一定的保护作用，作用机制可能与其抑制了兴奋性氨基酸毒性有关。
Conclusion Baicalin possesses protective effect on OGD injury in rat brain slices, which is related to its inhibitory effect on excitatory amino acids toxicity.
结论黄芩苷对大鼠脑片缺氧缺糖性损伤具有一定的保护作用，作用机制可能与其抑制了兴奋性氨基酸毒性有关。
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