The inhibitor for NO or for MMPs significantly reduced the LSEC IRI, suggesting that NO and MMPs are important mediators in the LSEC IRI.
应用一氧化氮和基质蛋白酶的抑制剂明显降低肝窦内皮细胞的损伤，表明一氧化氮和基质蛋白酶在缺血-再灌注损伤中是重要的介导物。
Objective:To investigate the role of granulocyte and adhesion molecules in the ischemia reperfusion injury of human liver sinusoidal endothelial cells(LSEC).
目的：探讨粒细胞和粘附分子在肝窦内皮细胞（LSEC）缺血再灌注损伤中的作用。
Objective:To investigate the role of granulocyte and adhesion molecules in the ischemia reperfusion injury of human liver sinusoidal endothelial cells(LSEC).
目的：探讨粒细胞和粘附分子在肝窦内皮细胞（LSEC）缺血再灌注损伤中的作用。
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