• These abnormal LPL cells produce large amounts of the protein immunoglobulin and macroglobulin.

    这些异常的LPL细胞产生大量的蛋白免疫球蛋白和巨球蛋白。

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  • But Marc Hamilton, Ph.D., one of Katzmarkzyk's colleagues, suspects it has to do with an enzyme called lipoprotein lipase (LPL), which breaks down fat in the bloodstream and turns it into energy.

    但是,凯茨玛德扎克的一个同事,马克·汉密尔顿博士怀疑,与一种叫脂蛋白脂肪酶(LPL)的酶有关,它能分解血管内的脂肪,将之转化为能量。

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  • Objective To explore the association of LPL gene polymorphisms with CHD.

    目的探讨脂蛋白脂酶(LPL)基因多态性与冠心病的关系。

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  • Lipoprotein Lipase (LPL) plays an important role in plasma lipoprotein metabolism.

    脂蛋白脂肪酶(LPL)在血浆脂蛋白的代谢中发挥了重要的作用。

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  • The first case of familial lipoprotein lipase (LPL) deficiency in China is re-ported.

    本文报道了我国首例家族性脂蛋白脂酶缺乏症。

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  • But the relationship between the LPL gene and hypertension was controversial among Caucasian populations.

    但是在白种人的研究中,LPL基因与高血压的关系尚存在争议。

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  • LPL possibly has a significant role in the pathogenesis of the case through participating in lipids abnormal metabolism.

    脂蛋白脂酶可能通过参与子痫前期高血脂异常代谢对子痫前期发病起着重要作用。

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  • Here we review the structure, function, expression regulation of the LPL gene along with its association with complex diseases.

    文章综述了LPL基因的结构、功能、表达调控以及与复杂疾病的关联研究进展。

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  • Conclusion: Overexpression of LPL in balloon injured carotid artery of rabbits can promote early atherosclerotic lesion formation.

    结论:LPL在兔动脉内膜损伤的血管壁局部高表达能够促进动脉粥样硬化早期病变的形成。

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  • The tissue-specific changes in LPL activity can be determined by the different rate of LPL gene transcription in different tissue.

    LPL活性调节的这种组织特异性主要是由于不同组织lpl基因转录速率不同所决定的。

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  • Analysis of amino acid composition of LPL shows that it has a high content of acidic amino acid, a few of basic amino acid but no Arg.

    氨基酸组成分析表明,该凝集素富含酸性氨基酸,而碱性氨基酸含量较少,不含精氨酸。

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  • Recent studies show that LPL activity is increased in skeletal muscle at the same time it is reduced in adipose tissue after acute exercise.

    大多数研究表明急性运动致骨骼肌脂蛋白脂酶(LPL)活性增强,脂肪组织LPL活性下降。

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  • Macrophages constitutively synthesize LPL, which may contribute to lipid accumulation within the arterial wall, promoting the atherogenic process.

    血管壁中巨噬细胞产生脂蛋白脂酶,使脂质积聚于动脉壁内,促进动脉粥样硬化进展。

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  • Results The levels of LPL mRNA in cardiac myocytes of diabetic group increased, but though BMOV treatment the levels of LPL mRNA decreased to control levels.

    结果糖尿病大鼠心肌脂蛋白脂酶mRNA是升高的,经BMOV治疗后心肌细胞脂蛋白脂酶m RNA表达可降低,基本恢复到正常对照水平。

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  • The above results indicated that human serum VLDL after hydrolysis with LPL in vitro have undergone significant changes in composition, structure and morphology.

    这些结果说明人血清VLDL经LPL分解代谢后,其结构,形态和组分均发生了明显的变化。

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  • The relationship between lipoprotein lipase (LPL) gene polymorphism and atherosclerotic diseases or stroke has become a topic of general interest in recent years.

    脂蛋白脂酶(LPL)基因多态性与动脉粥样硬化性疾病或卒中的关系成为近年来的研究热点。

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  • Among women with at least one copy of the minor allele at the LPL SNP, there was a reduced risk of mi only in active women, and not in those who exercised less frequently.

    在至少有一个LPL单核苷酸多态性等位基因的妇女中,仅在体力活动较多的女性中可减少心肌梗死的风险,而在那些体力活动较少的女性中未见此差异。

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  • Objective To study the expression of lipoprotein lipase(LPL) in human glomerular mesangial cells and the effect of very low-density lipoprotein(VLDL) on the expression of LPL.

    目的探讨人肾小球系膜细胞脂蛋白脂酶(LPL)的表达情况以及极低密度脂蛋白(VLDL)对其表达的影响。

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  • Moreover, decreasing LPL activity in adipose tissue and preventing excess lipids accumulation in tissues may contribute to the ability of FSH to maintain lipid and glucose homeostasis.

    此外,FSH能降低脂肪组织LPL活性,减少脂质在组织中的积累,也可能是FSH维持糖、脂代谢稳定的一个重要原因。

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  • From the research in the abdominal obesity, we know it can be caused by many factors, such as the gene, age, energy intake, lipoprotein lipase (LPL) reactive and the lipolysis reactive.

    腹型肥胖的发生与遗传、年龄、能量摄入、脂蛋白脂酶(LPL)活性及脂解活性等多种因素相关;

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  • The mono-clone cells of LPL/WM infiltrated other organs, with 12 cases of bone marrow infiltration, 5 of splenomegaly, 4 of lymphadenectasis, 1 of hepatomegaly, 1 of pulmonary infiltration.

    发病时多有脏器及血液系统受累,其中骨髓受累12例,脾脏受累5例,淋巴结受累4例,肝脏受累1例,肺部受累1例。

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  • Objective To explore the gene expression of hormone sensitive lipase (HSL) and lipoprotein lipase (LPL) between obesity-prone (op) and obesity-resistant (or) rats induced by a high-fat diet.

    目的探讨高脂饮食诱导肥胖易感(OP)大鼠和肥胖抵抗(OR)大鼠,激素敏感性脂肪酶(HSL)和脂蛋白脂肪酶(LPL)基因表达的差别。

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  • CONCLUSION: Taizhi "an capsule can decrease the hyperlipidemia. Its mechanism is related to the regulation of lipid metabolism and improvement of the concentration of LPL (lipoprotein lipase)."

    结论:泰脂安对高脂模型大鼠有较好的降脂作用,其降脂作用机制可能与泰脂安升高血清中的脂蛋白脂肪酶含量有关。

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  • But Marc Hamilton, Ph. D., one of Katzmarkzyk's colleagues, suspects it has to do with an enzyme called lipoprotein lipase (LPL), which breaks down fat in the bloodstream and turns it into energy.

    但是,凯茨玛德扎克的一个同事,马克·汉密尔顿博士怀疑,与一种叫脂蛋白脂肪酶(LPL)的酶有关,它能分解血管内的脂肪,将之转化为能量。

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  • But Marc Hamilton, Ph. D., one of Katzmarkzyk's colleagues, suspects it has to do with an enzyme called lipoprotein lipase (LPL), which breaks down fat in the bloodstream and turns it into energy.

    但是,凯茨玛德扎克的一个同事,马克·汉密尔顿博士怀疑,与一种叫脂蛋白脂肪酶(LPL)的酶有关,它能分解血管内的脂肪,将之转化为能量。

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