Objective: To demonstrate changes of KGF in premature rats with hyperoxia-induced CLD.
目的探讨高氧致CLD早产大鼠KGF表达的变化规律。
Objective to investigate the expression of insulin-like growth factor-binding protein-5 (IGFBP-5) in full-term rats with hyperoxia-induced chronic lung diseases (CLD) and its mechanism.
目的研究胰岛素样生长因子结合蛋白5 (IGFBP - 5)在高体积分数氧致新生大鼠慢性肺疾病(CLD)中的表达及作用机制。
Objective To study the effects of hyperoxia on ventilator-induced lung injury(VILI) and the protection of dexamethasone.
目的观察高浓度氧对通气机所致肺损伤的影响，以及地塞米松的防护作用。
Objective: To discuss the protect effect of Urokinase injection and Tan re-qing injection on lung injury induced by Hyperoxia in newborn rats.
目的：探讨注射用尿激酶、痰热清注射液对新生大鼠高氧肺损伤的影响。
Conclusion: the phosphorylation of RB protein can be induced by hyperoxia. Excessive proliferation of LF leads the pulmonary interstitial fibrosis.
结论：高氧可能导致RB蛋白磷酸化(RB灭活)，LF过度增殖最终导致肺间质纤维化。
Objective: To discuss the protect effect of Tan Re-Qing injection on lung injury induced by hyperoxia in newborn rats.
目的：探讨痰热清注射液对新生大鼠高氧肺损伤的影响。
The proteasome inhibitor MG-132 can reduce the lung injury induced by hyperoxia and inhibit P38MAPK signaling pathway.
蛋白酶体抑制剂MG- 132可以减轻高氧引起的肺损伤，可能对p 38 MAPK信号通路有抑制作用。
Ojective To establish a rat model of hyperoxia induced acute lung injury.
目的建立高氧性急性肺损伤大鼠模型并进行评价。
Ojective To establish a rat model of hyperoxia induced acute lung injury.
目的建立高氧性急性肺损伤大鼠模型并进行评价。
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