• Fine fibrils of proplastid were observed in electronic less dense region.

    在电子密度低的区域能观察到前质体的纤维丝。

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  • In contrast, insulin fibrils and lysozyme fibrils induced strongly hemolysis of the cells.

    相反,胰岛素纤维和鸡卵清溶菌酶纤维却能够引起红细胞的溶血。

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  • The spindle-knots have a rough surface, because the fibrils within them are randomly interweaved.

    纺锤节表面粗糙,因为里面的纤维是随机交织在一起的。

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  • The collagen fibrils had significant hyperplasia, and false liver acinus formed in liver of ILD.

    ILD组肝内胶原纤维明显增生,假小叶形成。

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  • The microtubular fibrils fibrils in the ultrastructure might be a concomitant phenomenon of mult...

    超微结构中的微管状纤维物可能是多种系统性疾病的伴随现象。

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  • Tendons are composed of highly aligned collagen fibrils and elongated fibroblasts known as tenocytes.

    肌腱组织的主要组成是胞外基质中的胶原纤维和镶嵌在胶原纤维内的肌腱细胞。

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  • The number of twisting collagenous fibrils closely correlated with the degree of fibrous degeneration.

    斑块状扭结的数量与胶原原纤维的退变程度成正比。

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  • With SEM, fibrils could be seen, it was difficult, however, to further observe the fine structures in fibrils.

    结果表明,SEM只能观察到纤丝在棉花纤维表面的排列,但很难进一步观察到纤丝的精细结构。

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  • By electron microscopy, amyloid is composed of a "beta-pleated sheet" of fibrils, seen here as irregular grey material.

    电镜,淀粉样物质是由原纤维的β-折叠组成。可见不规则的灰色物质。

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  • Cuticular protein and chitin self-assemble into regularly arranged fibrils, which is the fundamental structure of cuticle.

    表皮蛋白和几丁质通过自我组装形成复合物的超高级结构,这种结构是昆虫表皮行使功能的基础。

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  • Collagenase is one kind of enzymes that can degrade native helical collagen fibrils or glutin but not degrade others proteins.

    胶原酶是一种特异降解胶原螺旋区或明胶,而不作用于其它蛋白质底物的酶类。

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  • Due to the breakage of the LCP micro-fibrils, the strength of the drawn PP/LCP fibers is lower than that of the pure PP fibers.

    在牵伸过程中由于液晶高分子微纤维的断裂而使共混纤维的强度低于纯聚丙烯纤维;

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  • Except that the fibrils occurred loose, the structure of DSPP treatment had no distinct alteration and was similar with control.

    DSPP处理对胶原纤维的影响不大,结构基本上与对照接近,只是稍稍发生松动。

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  • Furthermore, it is revealed that the fracture of fibre results from the longitudinal sliding displacement of fibrils or microfibrils.

    还发现聚乙烯纤维在单向拉伸下的断裂是通过细纤维或细纤维束的相互滑移而实现的。

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  • It is essential to elucidate the cytotoxic mechanism of amyloid fibrils for developing novel medicines for the treatment of amyloid diseases.

    阐明淀粉样纤维的毒性机制对于研制用于治疗淀粉样疾病的药物显得异常重要。

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  • The surface of BC membranes was porous network structure observed by SEM, and the micro-fibrils of BC were secreted from the eyelet of cell wall.

    扫描电镜下可见细菌纤维素膜呈疏松的网状结构,纤维素微纤丝从菌体胞壁小孔中分泌出来;

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  • During bone resorption, the inorganic components of the bone matrix were first dissolved and resorbed leaving behind rough and irregular collagen fibrils.

    骨吸收过程中,无机盐可以先吸收,残剩粗糙不规则的胶原纤丝。

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  • Electron microscopy reveals the characteristic fibrils of amyloid and in early cases this technique is occasionally essential for the diagnosis to be made.

    电子显微镜能显示出淀粉样蛋白纤维的特征,在早期的病例中这项技术有时是诊断所必需的。

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  • The main composition of extracellular matrix of compact bone, collagen fibrils and minerals, were preserved in the natural position and structure characteristic.

    密质骨骨基质的主要成分胶原纤维和骨盐仍维持原有形态及结构特征,胶原纤维聚集、排列成规则的网状支架。

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  • The collagen fibrils in the dentine of aging rat enlarged in its diameter, and were denser in the pattern of their distribution and ran randomly in their direction.

    老化牙本质胶原原纤维的横纹模糊不清,数量增加,口径增粗,分布致密,走向随意;

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  • The structure of endothelial cells of capillaries was changed, the basal lamina became thicker and the pericapillary space became broader with a lot of collagen fibrils.

    毛细血管内皮细胞形态结构发生改变,基膜增厚,血管周隙明显增宽,内含大量的胶原原纤维。

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  • The average diameter of PA6 fibrils decreased with the increasing draw rate, and the diameter distribution of fibrils became narrow, the mechanical properties were improved.

    发现随牵引速度的提高,PA 6微纤的平均直径变小,尺寸分布更均匀,力学性能也随之提高。

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  • Osteoarthritis occurs when the fibrils are disrupted or lost - just like concrete without the steel, the cartilage becomes mechanically weak and susceptible to wear and tear.

    当原纤维遭到破坏或者缺失就像混凝土缺少钢筋一样,骨性关节炎就会发生。软骨的机械性变弱易于磨损和撕裂。

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  • But the precise structure of these fibres or 'fibrils' has remained a mystery for more than 40 years, so hindering any progress towards the development of potential therapies.

    但是这些纤维或者“原纤维”的精密结构保持它的神秘达40年之久,妨碍了可能的治疗方法研究的进展。

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  • Usually amyloid fibrils form and deposit in the vicinity of the target cells and accumulate on the cell membrane, triggering a cascade of cellular events which lead to apoptosis.

    通常情况下,淀粉样纤维在靶细胞的附近沉积,在细胞膜上聚集,引起细胞的连锁反应而导致细胞的死亡。

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  • Now, a team from the University of Manchester has used sophisticated electron microscope techniques to uncover the molecular structure of the thinner of the two types of collagen fibrils.

    现在,来自曼彻斯特大学的一个研究小组利用高超的电子显微镜技术揭示了这两种胶原纤维的薄层的分子结构。

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  • ConclusionThe intra-retinal collagen fibrils penetrating the ILL and mixing with the cortical vitreous fibrils appears to underlie the slow adhesion of the vitreoretinal interface with aging.

    结论随年龄增长来自视网膜内的胶原纤维逐渐穿透ill与玻璃体皮质纤维缠绕结合,使基底部玻璃体视网膜界面粘连延伸。

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  • ConclusionThe intra-retinal collagen fibrils penetrating the ILL and mixing with the cortical vitreous fibrils appears to underlie the slow adhesion of the vitreoretinal interface with aging.

    结论随年龄增长来自视网膜内的胶原纤维逐渐穿透ill与玻璃体皮质纤维缠绕结合,使基底部玻璃体视网膜界面粘连延伸。

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