结论LPS致宫内感染可造成胎鼠脑损伤。
Conclusions Fetal rat brain damage is made by LPS-induced intrauterine infection.
然后研究者把发育中的胎鼠的脑暴露于损伤。
Then the researchers exposed the brains of the developing fetuses to injury.
目的：探讨诊断超声对胎鼠内耳细胞是否构成刺激。
Objective: To evaluate whether diagnostic ultrasound should stimulate inner ear of fetal rats.
死胎率和畸胎率均有所下降，而胎鼠的生长发育受到干扰。
Both the rate of dead fetuses and of the monstra were decreased but the development of the fetuses was disturbed.
实验组胎鼠的身长、尾长和体重等发育指标均有显著的降低。
The mean body weight, average body length, tail length significantly decreased.
结论维甲酸有较强的胎鼠骨骼致畸作用，这种作用是多部位的。
Conclusion Retinoic acid has a strong and multi-regional teratogenic effect on the skeleton of fetal rat.
本文研究了敌草快和肾上腺皮质激素对胎鼠动脉管的收缩作用。
The toxic effects of diquat and adrenocortical hormone on ductus arteriosus of rat fetus were studied in this paper.
本文研究了敌草快和肾上腺皮质激素对胎鼠动脉管的收缩作用。
The present experiments were conducted to study the relationship of contraction of ductus arteriosus of fetuses induced by diquat with adrenocortical hormone and its receptor.
目的：从分子水平探讨叶酸缺乏（FD）对胎鼠脑发育的影响。
Objective: To verify the effect of maternal folate deficiency (FD)on fetal rat brain development, and the change of DNA methylation.
目的观察胎鼠发育过程中脑内神经干细胞增殖、迁移和分化特点。
Objective To evaluate the characteristics of proliferation, migration and differentiation of neural stem cells in the brain of rat embryo during development.
结果表明体外培养的胎鼠海马神经元的网络活动具有温度敏感性。
These showed that the activity of hippocampal neuronal networks is sensitive to the temperature.
结论BMP4可体外上调胎鼠海马及大脑皮质神经元的胆碱能表达。
Conclusion BMP4 may in vitro up-regulate the cholinergic expression of the neurons in hippocampus and cerebral cortex of embryo rats.
方法胎鼠头盖骨成骨细胞在含有不同浓度孕酮的培养基中培养约20天。
Methods Fetal rat calvarial osteoblasts were cultured in the medium with different concentrations of progesterone for nearly 20 days.
研究发现15日龄的胎鼠卵巢内即存在P450（SCC）的免疫染色；
The results showed that P450 (SCC) was located in the mouse ovary of 15-day pregnancy;
期末血清叶酸、胎鼠血清叶酸及胎鼠脑组织DNA甲基化水平作相关分析。
Correlation analysis was processed among serum folate concentration of dam and fetus and DNA methylation level in fetal brain.
用骨骼染色法观察胎鼠有无骨骼畸形，内脏切片法观察胎鼠内脏畸形情况。
The fetus skeletal development and visceral abnormality were observed by skeletal staining and visceral section.
目的研究胎鼠胃上皮细胞增殖、凋亡的时空规律及其与胃形态发生的关系。
Objective to study the spatiotemporal distributions of epithelial proliferation and apoptosis, and the correlation of morphogenesis with them in developing stomach.
结论：与两对照组相比，胎鼠肢芽提取液呈现了显著的运动神经营养活性。
Conclusions: the limb bud extract showed marked motoneuron neurotrophic activities compared with the two controls.
结果显示，复合作用对胚胎的致畸毒性有所减轻，但对胎鼠的损伤效应不一。
The results indicate that there was a decrease in teratogenic embryotoxicity of the combined treatment than the single treatment, but the damage effects on the fetuses of the rats is inconsistent.
结论：胎鼠听囊细胞在体外自然培养条件下可以分化或者增殖为未成熟毛细胞。
CONCLUSION: in natural culture condition in vitro, otocyst cells of fetal rats can differentiate or proliferate to immature hair cells.
从胎鼠中取出的肺细胞被注入这个装置系统中，同时还有帮助细胞生长的营养物质。
Lung cells taken from rat fetuses were injected into the setup, along with nutrients that helped the cells grow.
目的了解蚂蚁粉对宫内发育迟缓（IU GR）胎鼠的大脑，肝脏和胎盘的作用。
Objective To investigate the effect of ant powder on the brain, liver and placenta of fetal rat with IUGR.
目的：研究宫内生长受限(FGR)胎鼠脑发育及生后远期学习记忆能力的变化。
Objective: To study the changes of cerebral development and learning and memory ability of fetal rats with intrauterine fetal growth restriction (FGR) after birth.
结论血清igfbp - 1作为生长抑制因子参与正常胎鼠发育及IUGR的发生。
Conclusions Serum IGFBP-1 participates in the growth of normal and IUGR rat fetuses as an inhibiting factor.
目的探讨妊高征（PIH）大鼠颈交感神经干离断（TCST）对胎鼠脑的保护作用。
Objective To investigate the protective effect of transection of cervical sympathetic trunk (TCST)on the fetal brain in rats with pregnancy-induced hypertension(PIH).
结论：孕酮对离体胎鼠头盖骨成骨细胞的分化具有多重促进效果，但对细胞的增殖无影响。
Conclusion: Progesterone has a multi stimulating effect on the differentiation of fetal rat calvarial osteoblast, but no effect on cell proliferation.
结论：FGR胎鼠在宫内即可有神经系统损伤，并对出生后远期的学习记忆能力产生影响。
Conclusion: FGR may induce the nervous system damage in the intrauterine fetal rats and further affect the learning and memory ability after birth.
结论孕酮从基因水平调节离体胎鼠成骨细胞骨钙素的生成，这种调节呈现时间及剂量依赖。
Conclusion progesterone was involved directly in osteocalcin production by regulating gene expression and this effect of progesterone was time and dose dependent.
结论孕酮从基因水平调节离体胎鼠成骨细胞骨钙素的生成，这种调节呈现时间及剂量依赖。
Conclusion progesterone was involved directly in osteocalcin production by regulating gene expression and this effect of progesterone was time and dose dependent.
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