结论：纤维蛋白血凝块表型是疾病严重程度及卒中后长期死亡率的预测因素。
Conclusions: Fibrin clot phenotypes are indicators of both severity of disease and long term post-stroke mortality.
采用发射光谱法分析了急型克山病人的去纤维蛋白血中的铜、镁、锰和锌的浓度。
The concentration of copper, magnesium, manganese and zinc in thedefibrinated blood of patients with acute Ke-shan disease has been determinated by emission spectrometry.
小纤维蛋白血栓可在脑、心脏、肺、肾以及其它器官的小动脉中形成，引起组织缺血。
Small fibrin thrombi can form in small arteries of brain, heart, lungs, kidneys, and other organs to produce ischemic tissue damage.
表明纤维蛋白原结合唾液酸的水平可作为诊断肝硬化获得性异常纤维蛋白血症的理想指标。
Polymerization of fibrin monomer was inverse ratio with fibrinogen bound sialic acid. It suggests that fibrinogen boud sialic acid levels may be diagnostic index acquired dysfibrinogenaemia.
表明纤维蛋白原结合唾液酸的水平可作为诊断肝硬化获得性异常纤维蛋白血症的理想指标。
Polymerization of fibrin monomer was inverse ratio with fibrinogen bound sialic acid. It suggests that fibrinogen boud sialic acid levels may be diagnostic index acquired dysfibrinogenaemia.
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