模型组大鼠心肌细胞结构出现明显的病理改变。
The obvious pathological changes of cardiomyocyte structure were found in model group.
结论：心肌肽素通过抑制心肌细胞凋亡，减轻心肌细胞结构损伤发挥对大鼠心肌缺血再灌注损伤的保护作用。
CONCLUSION: Cardiomyopeptidin plays a role in protecting myocardial ischemia-reperfusion injury in rats by inhibiting myocardial apoptosis and alleviating myocardial structural lesions.
它们对于心肌细胞起结构支持作用，在生长和病理条件下，负责细胞外基质的合成。
They provide structural support for cardiac myocytes and are responsible for extracellular matrix synthesis in the heart during growth and pathophysiological conditions.
目的：研究诺迪康对心肌细胞超微结构的影响以探讨该药对心室肌重构的作用与心功能增强的关系。
Purpose:To explore the effect of rhodiola on myocardial ultramicrostructure and the relationship between myocardial restructure and cardiac function under the drug.
HCY2基因可引起心脏结构紊乱、心内膜垫缺损和抑制心室心房发育，并可损害心肌细胞的超微结构。
Under light and electron microscope, it was seen that HCY 2 gene resulted in structure disturbance, endocardial cushion defect and hypoplasia of heart.
分别在术后1、2、3个月时采用透射电镜观察缺血心肌细胞超微结构的改变情况。
The changes of ultrastructure of ischemic myocardium were observed with electrical microscope at 1,2,3 months after the operation.
本实验对大鼠心房心肌细胞的肌浆网与闰盘间的关系进行了超微结构观察。
The ultrastructure of the relationship between the sarcoplasmic (SR) and the intercalated disk in the atrial myocardial cells of the heart is studied.
我们的研究证实胆固醇可以调节离子通道亚膜池使之随时补充心肌细胞表面膜结构。
Here we show that cholesterol regulates the submembrane pool of ion channels readily available for recruitment into the surface membranes of heart cells.
目的：研究4种心肌保护方法对心肌细胞质膜蛋白结构和功能的影响，评价其心肌保护效果。
Objective: to investigate the effects of 4 kinds of myocardial protection methods on the myocardial membrane-proteins (MMPs), and to evaluate their cardioprotective effects.
目的：探讨法鲁四联症心肌细胞光电镜结构的变化与其功能的关系。
Objective: we studied the relationship between structural alteration of the myocyte and function under light and electron microscope.
目的：观察心虚证对心肌细胞微细结构及超微结构的影响。
Objective: To observe the effect of heart deficiency syndrome on microstructure and ultra-microstructure of cardiac muscle cells.
细胞的形态和结构介于P细胞和心肌细胞之间。 最后对窦房结内各细胞的功能特点加以讨论。
The shape and structure of T cell is between P and myocardial cell, and situated on the periphery of node.
目的：研究心衰时心肌细胞及亚细胞结构的形态学改变。
Objective: To study the morphological changes of cardiac myocytes and the subcellular structure of overload hart failure.
心肌细胞超微结构观察结果：糖尿病组心肌组织内可见凋亡的心肌细胞。
Results of ultrastructure of cardiac myocyte: Apoptosis could be observed in myocardial cell in diabetic group.
结果(1)he法：uc组大鼠心肌组织结构正常，IH组心肌灶性变性，心肌细胞肿胀，间质中有炎性细胞浸润。
Results (1) he method: the changes of vessel in heart, lung and kidney in UC group were not found, while the rat in IH group were found for myocardial cell swelling and inflammatory cell infiltration.
目的：观察低硒喂养对大鼠心肌组织脂质过氧化物、心肌细胞超微结构及细胞凋亡的影响。
AIM: to investigate the effects of low-selenium diet on rats 'myocardial lipid peroxidize and apoptosis.
心肌胶原是心肌间质的重要组成部分，与心肌细胞共同维护心脏结构和功能的完整。
Myocardial collagen is an important part of myocardial interstitium. It maintains constitional and functional integrity of heart with cardial muscle cell.
观察两组患者心脏电生理指标、血流动力学恢复、正性肌力药物用量、心肌细胞超微结构和心肌酶浓度变化等。
Cardiac electrophysiologic data, recovery of hemodynamics, inotropic medicine dosage, myocardial ultrastructural changes and concentration of myocardial enzymes were assessed.
方法将DM大鼠随机分为西洛他唑治疗组和DM组，用药8周后检测两组血糖、体质量、糖化血红蛋白、心脏重量指数，观察其心肌细胞超微结构变化。
DM rats were randomly divided into cilostazol group and DM group. 8 weeks after treatment, blood glucose, body weight, HbA1c, cardiac weight index and ultrastructure of myocardiocyt were observed.
方法取SHR大鼠的右心房作透射电镜观察，分析心肌细胞的超微结构。
Methods: The transmission electron microscopy was used to study the ultrastructure of the cardiac myocytes in right atrium.
缺氧、烧伤血清可引起心肌细胞骨架结构破坏及重构，网络结构消失，细胞完整性破坏；
Hypoxia and burned serum could result in the myocardial cytoskeleton damage, and pathological remodeling, therefore cytoskeletal network disappeared, and cell integrity damaged.
在晚期心力衰竭的心肌细胞中，横管结构及其规则横纹排列模式已基本消失。
Myocytes from the failing hearts lose the majority oft-tubules and their regular striated pattern.
结果：分化细胞中可见节律性收缩的细胞，经超微结构分析和免疫细胞学鉴定为心肌细胞。
RESULTS:Rhythmically contracting cells were observed among differentiated cells, which were proved to be cardiomyocytes with electron microscope and immunocytochemistry.
心肌细胞超微结构表现为损伤减轻。
结论：骨髓间质干细胞分化为心肌细胞过程中，随培养时间延长及细胞密度增加逐渐形成闰盘样结构。
CONCLUSION: During the process of BMMSCs differentiating into cardiomyocytes, intercalated disc-like structure is gradually formed with increase in culture time and cell density.
心肌细胞核染色质结构接近正常；
Nucleolus chromatin structure of cardiomyocytes was close to normal;
心肌细胞核染色质结构接近正常；
Nucleolus chromatin structure of cardiomyocytes was close to normal;
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