结论流式细胞术在循环内皮细胞检测中优于手工法。
Conclusion the efficiency of flow cytometry was superior to manual counting in measuring circulating endothelial cells.
循环内皮细胞的数量与年龄、循环祖细胞的数量没有关系。
检测16例呼吸衰竭（呼衰）患儿血中循环内皮细胞（CEC）含量。
The circulating endothelial cells (CEC) of 15 healthy children and 16 patients with respiratory failure were counted respectively.
关于继发于先心病的肺高压患者循环内皮细胞水平没有数据可以参考。
No data are available on CEC levels in PAH secondary to CHD.
方法随机选择血常规标本20例，用流式细胞术测定外周血循环内皮细胞，并与手工法比较。
Methods Flow cytometry and manual counting were applied to measure circulating endothelial cells in 20 blood routine samples.
目的了解系统性红斑狼疮(SLE)患者外周血循环内皮细胞(cec)的数量及其临床意义。
Objective to investigate the number of CEC in peripheral blood of patients with systemic lupus erythematosus (SLE) and its clinical significance.
以循环内皮细胞(CEC)作为血管内皮损伤的指示物，血浆内皮素(ET - 1)反映血管内皮的功能变化。
Circulating endothelial cells (CEC) were used as an endothelial injury indicator and the plasma level of endothelin (ET-1) as the functional marker of the vascular endothelial cells.
循环内皮细胞的数量在不可逆肺高压组显著高于可逆性肺高压组和对照组，然而这三个亚组的循环祖细胞数量却没有差异。
CEC Numbers were significantly higher in patients with irreversible PAH than in patients with reversible PAH and control subjects, whereas CPC Numbers did not differ among the 3 subgroups.
本文以原代培养的人脐静脉内皮细胞（HUVEC）为材料，研究了肿瘤坏死因子（TNF）对HUVEC的损伤作用及循环内皮细胞（CEC）的形成机制。
The effects of tumor necrosis factor (TNF) on cultured human umbilical vein endothelial cells (HUVEC) and the mechanism of circulating endothelial cell (CEC) formation had been studied.
目的：观察多器官功能障碍综合征(MODS)大鼠肺组织中循环内皮细胞(CEC)以及细胞间黏附分子(ICAM - 1)和血管细胞黏附分子(VCAM - 1)的表达，并探讨它们之间的相互关系。
Objective: to observe the MODS rat lung tissue's CEC, as well as adhesion molecules ICAM-1 and VCAM-1 expression changes, and to investigate the relationship between them.
研究人员让14名身体健康的志愿者分别饮用绿茶、稀释的咖啡和热水，然后对他们的循环系统内皮细胞的机能进行检测。
The researchers gave 14 healthy volunteers either green tea, diluted caffeine or hot water and then measured the function of endothelial cells lining the circulatory system.
目的探讨体外循环(CPB)期间低温改良低钾右旋糖酐(LPD)溶液肺动脉灌注对CPB中肺血管内皮细胞损伤的保护作用。
Objective To test the effect of pulmonary artery perfusion with modified low-potassium dextran (LPD) solution on lung vascular endothelial cell injury during cardiopulmonary bypass (CPB).
目的研究门静脉高压症病人肝组织内血管及肝窦壁内皮细胞的凋亡及其对肝微循环的影响。
Objective To investigate the intrahepatic vascular and hepatic sinusoidal endothelial cell (SEC) apoptosis and its influence on hepatic microcirculation in patients with portal hypertension.
目的探讨急性胰腺炎(AP)胰腺微循环中血小板内皮细胞粘附分子1 (PECAM 1)表达的变化规律。
Objective To investigate the changes of platelet endothelial cell adhesion molecule 1 (PECAM 1) expression in leukocytes in pancreatic microcirculation of experimental acute pancreatitis (AP).
结论体外循环可诱导炎症反应致内皮细胞损伤或激活，并可致血管通透性增高。
Conclusion CPB can induce activation damage of vascular endothelial cell, and increase vascular permeability.
血管内皮祖细胞是一类能循环、增殖并能直接分化为血管内皮细胞的前体细胞。
Endothelial progenitor cell (EPC) is a subtype of progenitor cells that can circulate, proliferate and have the ability to differentiate into mature endothelial cells.
目的：探讨改善微循环对周围神经嵌压性损害血管内皮细胞生长因子(VEGF)表达和病理改变的影响。
Aim: to study the influences on expression of vascular endothelium growth factor (VEGF) and pathologic changes by improving microcirculation following crushing lesion of sciatic nerve in rats.
结论NAC可以抑制CPB血清所致的培养血管内皮细胞ICAM - 1表达增加。因此NAC对体外循环后全身炎症反应可能有保护作用。
Conclusion NAC could inhibit ICAM-1 expression on cultured endothelial cells, and might play a role in attenuating systemic inflammatory response syndrome induced by CPB.
本研究研发了一个新模型显示，中性粒细胞通过物理作用和锚定循环肿瘤细胞到内皮细胞上来调节肺转移的进展。
A novel model has been developed in this study showing that neutrophils regulate lung metastasis development through physical interaction and anchoring of circulating tumor cells to endothelium.
虽然越来越多的实验表明白细胞以及内皮细胞在体外循环中所导致的炎症反应中占据着重要地位，但炎症反应发生的确切机理尚不明确。
Despite the increasing evidence for the role of neutrophils in inflammation subsequent to bypass, the exact pathophysiology of the damage to this remains poorly understood.
虽然越来越多的实验表明白细胞以及内皮细胞在体外循环中所导致的炎症反应中占据着重要地位，但炎症反应发生的确切机理尚不明确。
Despite the increasing evidence for the role of neutrophils in inflammation subsequent to bypass, the exact pathophysiology of the damage to this remains poorly understood.
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