研究人员发现，选择性杀死一到两种免疫应答细胞不会对小鼠抵抗癌症产生不良影响。
The researchers discovered that selectively killing off one or two of the responding cell types didn't affect how well a cancer-resistant mouse fended off the disease.
从应答方面来讲，动物的免疫防御系统又划分为先天性免疫细胞及分子和适应性免疫细胞及分子。
Animal immunizing and defending system is also divided into geneogenous immunizing cells and molecules & adaptive immunizing cells and molecules on response.
许多研究者认为t细胞应答能限制疾病的严重程度，但不能预防疾病。
Many researchers think a T-cell response would limit disease severity but would not stop you falling ill.
这个途径叫TOR，涉及细胞对营养物质的应答。
Called TOR, this pathway is involved in a cell's response to nutrients.
尽管如此，这种激起细胞介导的对流感的免疫应答的想法仍存在争议，部分是因为大规模生产t细胞比抗体更耗时。
The idea of provoking a cell-mediated response to flu is controversial, though, in part because it takes longer to start mass-producing T-cells than antibodies.
被选中的寄主细胞是一种被叫做T细胞的免疫系统细胞，具体而言，他们是辅助T细胞[注一]，它将应答免疫系统对例如病毒等病原体的反应。
The host cells in question are immune-system cells called T-cells. Specifically, they are "helper" T-cells, which orchestrate the immune system's response to pathogens such as viruses.
研究人员认为，骨髓干细胞可以减少呼吸道的炎症反应，减轻免疫系统对严重的哮喘的免疫应答。
The researchers believe the bone marrow stem cells reduce inflammation in the airways, the immune system's normal response to severe cases of asthma.
肺泡巨噬细胞(AM)被GM - SCF强化，这是先天免疫应答的重要部分，已知在动物模型有助宿主防御流感病毒感染。
Alveolar macrophages (AM), which are enhanced by GM-SCF, are an essential piece of the innate immune response and are known to contribute to host defense against flu infections in animal models.
本文综述鼻腔免疫应答的细胞和分子机理、抗原释放系统及佐剂等方面的最新研究进展。
We summarize the recent advances of the cellular and molecular mechanism, the delivery system of antigen and adjuvant of intranasal immunization.
我们研发的疫苗很多只是形成新的抗体，但是抗体记忆不同于T细胞记忆，因为它们是应答产生的各种蛋白质。
A lot of our vaccines only develop new antibodies and antibody memory is different from t cell memory because of the types of proteins they respond to.
当细胞积累了大量的错误折叠蛋白后，细胞的报警系统被打开，细胞应激应答被激活。
In response to the accumulation of misfolded proteins, the cellular alarm systems are switched on and stress responses are activated.
而且不同的盐激时间还出现不同的差异蛋白，说明其细胞内存在复杂的应答盐激的蛋白质网络调控。
In addition, the appearance of differential displayed proteins responding to different salt shock periods was also demonstrated, illuminating that complex network of regulation exist in cell.
这种细胞活性很像一次失控的免疫应答反应。
The cellular activity resembles an immune response that has spiraled out of control.
在应答缺血性心肌损伤时，脾单核细胞能动性增加，全体离开脾，在受伤的组织聚集，参与伤口愈合。
In response to ischemic myocardial injury, splenic monocytes increase their motility, exit the spleen en masse, accumulate in injured tissue, and participate in wound healing.
异种细胞间的粘附在免疫应答等的生命活动中也起重要的作用。
Adhesion between heterogeneous cells also plays important roles in life activities, such as immune response etc.
每当机体对病原体产生应答时，这些同种免疫细胞弥散性分布于参与免疫应答过程中。
Every time you respond to a pathogen, these alloimmune cells are randomly sprinkled throughout all of your immune responses.
氯离子参与生物体多种功能，如细胞免疫应答、细胞迁移、增生、分化和凋亡。
Chloridion participated in various kinds of biological function, such as cellular immunologic response, cell migration, cellular proliferation and differentiation, apoptosis.
研究者们发现受损皮肤细胞能分泌TSLP(胸腺间质的淋巴细胞生素)，这是一种能引起强免疫应答的复合物。
The researchers found that cells in damaged skin can secrete TSLP (thymic stromal lymphopoietin), a compound capable of eliciting a powerful immune response.
目的：研究雷洛昔芬对血管内皮细胞雌激素应答元件(ERE)转录活性的影响与雌激素受体(er)亚型的关系。
Objective: to investigate the effect of Raloxifene on the estrogen responsive element (ERE) transcription activity and its relationship with estrogen receptor subtypes in vascular endothelial cells.
该病毒破坏多种淋巴细胞，致使身体免疫应答抑制。
The virus destroys a subgroup of lymphocytes, resulting in suppression of the body's immune response.
结论东阿阿胶能解除或减轻肿瘤和放疗对免疫系统产生的抑制作用，有助于免疫细胞对肿瘤的应答。
CONCLUSION DHG can get rid of or relieve the immune system depression induced by tumor and radio-therapy and conduce to immunocyte response to tumor.
我希望你们记住的是，一些特定的细胞亚群受到刺激时，被激活细胞将引起特定的应答。
What I want you to remember is that specific sub-populations of cells get activated, the activation results in a specific response.
在病原、诱发因子和激素应答中是调节细胞程序性死亡的关键因子。
It is the key factor regulating programmed cell death in pathogen, evocator and hormone responses.
在病原、诱发因子和激素应答中是调节细胞程序性死亡的关键因子。
It is the key factor regulating programmed cell death in pathogen, evocator and hormone responses.
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