近年来,在PD CD 4基因对程序性细胞死亡的影响,以及对转化和蛋白质翻译的抑制作用的相互关系等方面进行了大量研究。
Quantity of studies of PDCD4 have been carried out about its effect on programmed cell death and correlationship with inhibition for transformation and protein translation in recently year.
在病原、诱发因子和激素应答中是调节细胞程序性死亡的关键因子。
It is the key factor regulating programmed cell death in pathogen, evocator and hormone responses.
应用DNA凝胶电冰、电镜观察和流式细胞仪分析,对加热诱导G422胶质瘤程序性细胞死亡的作用进行了研究。
Using DNA gel electrophoresis, electron microscopic examination and flow cytometric analysis, program cell death induced by hyperthermia in mouse G422 glioma in vivo was studied.
对于软骨和椎间盘的损伤和压缩导致细胞坏死或程序性细胞死亡已经明确了。
It is known that compressive or traumatic injuries to cartilage and intervertebral discs can result in cell death by necrosis or apoptosis.
当这个基因被失活时,植物就不能再调节程序性细胞死亡过程,从而导致整个叶子和植物的死亡。
When the gene is inactivated, the plant can no longer regulate PCD, leading to cell death throughout the leaf and plant.
程序性细胞死亡有两种,包括凋亡和自噬性死亡,二者具有不同的形态学特征。
There are two morphologically distinctive forms of programmed cell death, apoptosis and autophagic cell death.
细胞程序性死亡是植物发育和响应环境胁迫过程中的重要过程。
Programmed cell death (PCD) is a crucial process for plants during development and environmental stress.
本文就细胞程序性死亡在植物适应逆境中的重要作用进行了综述,以期对细胞程序性死亡的研究进一步深入和对植物适应逆境的潜力有新的认识。
Research progress for the process of PCD was summarized and role of PCD in plant adaptation to environmental stress was discussed. Research prospective was also put forward in this paper.
本文就细胞程序性死亡在植物适应逆境中的重要作用进行了综述,以期对细胞程序性死亡的研究进一步深入和对植物适应逆境的潜力有新的认识。
Research progress for the process of PCD was summarized and role of PCD in plant adaptation to environmental stress was discussed. Research prospective was also put forward in this paper.
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