Therefore, we hypothesized that sevoflurane influences cerebral autoregulation.
因此，我们假设七氟醚影响脑自动调节。
Therefore, we hypothesized that apoptosis is a significant mechanism of injury to duct epithelium.
因此，我们推测细胞凋亡是胆管上皮细胞损伤一个重要机制。
So we hypothesized that rejected children included highly obedient and manipulated children, highly aggressive children.
因此我们假设被拒绝的儿童包括高度顺从和容易受摆布的儿童，还有高度攻击性的儿童。
So we hypothesized that women would naturally be more leery of negatively framed information when evaluating a prospective mate.
因此我们推测女性在衡量未来的另一半时，对消极信息更加敏感。
By analyzing the spread function and development contrast curve, we hypothesized that the resolution limit of electron-beam lithography is primarily limited by developer-diffusion.
通过对电子束扩散函数与显影对比度的分析，本文认为电子束曝光的分辨极限与显影液在纳米尺度下的扩散限制有关。
We hypothesized that an uncemented femoral component is a risk factor for bleeding.
我们假设非骨水泥型股骨假体是出血的危险因素。
We hypothesized that such HIV suppression requires ACV phosphorylation by HHV kinases.
我们推测这种抑制作用依赖于人疱疹病毒酶导致的阿昔洛韦磷酸化。
We hypothesized that underlying key factors can be identified at the onset of the disease.
我们假设在疾病的开始就能识别这些基本的关键因子。
We hypothesized that germline variation contributes to MPN predisposition and phenotypic pleiotropy.
我们推测种系变异可促进MPN易感及表型多向性。
We hypothesized: Each achievement goal had a distinct effect from another goal on working memory and controlled retrieval.
研究的基本假设是：不同成就目标定向对工作记忆广度和工作记忆的控制性提取功能具有不同影响。
We hypothesized that TNF induces opposing inflammatory and remodeling responses in HF that are TNF-receptor (TNFR) specific.
我们的研究假设是TNF通过特异的TNF受体(TNFR)诱导相反的炎症和重构反应。
Objectives: We hypothesized that a standardized outpatient clopidogrel desensitization protocol would be safe and effective.
目的：我们假设门诊病人标准化氯吡格雷脱敏疗法是安全且有效的。
We hypothesized that alterations in cellular protein S-nitrosylation may contribute to hyperglycemia-induced endothelial dysfunction.
我们假设细胞蛋白s -亚硝基化的变化会引起高血糖介导的内皮功能障碍。
We hypothesized that a protein preload would reduce the postprandial glycemic excursion in type 2 diabetic patients by these mechanisms.
根据以上机制，我们认为餐前服用些许蛋白质可以降低2型糖尿病患者的餐后高血糖。
We hypothesized that combinatorial expression of neural-lineage-specific transcription factors could directly convert fibroblasts into neurons.
我们假设，联合表达神经系特异性转录因子可以直接将成纤维细胞转换成神经元细胞。
We hypothesized that the mouse molar has intrinsic potential to maintain the epithelial stem cell niche and assessed this potential by growth in vitro.
我们提出假说，即鼠类磨牙具有维持上皮干细胞龛的内在潜能，并通过体外实验来证明此假说。
Background: Adverse cardiac events are common after vascular surgery. We hypothesized that perioperative statin therapy would improve postoperative outcomes.
背景：不良心脏事件常见于血管手术后。我们猜测围术期氟伐他汀治疗会改善术后结局。
We hypothesized that the extent of edema has prognostic implications and that diffusion-weighted MR imaging (DWI) can help predict the progression to infarction.
本文将探讨水肿范围的预后意义，并通过MR弥散成像(DWI)来预测梗死的形成。
We hypothesized that patients receiving a collagen meniscus implant would have better clinical outcomes than patients treated with partial medial meniscectomy alone.
我们假设胶原半月板移植治疗的临床效果要好于单独行半月板部分切除术。
We hypothesized that fascial displacement in experimental compartments with impaired PP would be significantly greater than that in control compartments with normal baseline PP.
我们假设灌注压受损的实验组筋膜间室的筋膜移位程度明显大于灌注压保持正常基线状态的对照组筋膜间室。
We hypothesized that women with hot flashes would show reduced flow-mediated dilation and greater coronary artery and aortic calcification compared with women without hot flashes.
对比没有烘热症状的妇女，伴有烘热症状的妇女显示较低流量介导的扩张以及更严重的冠脉和主动脉钙化。
We hypothesized that lenient rate control is not inferior to strict rate control for preventing cardiovascular morbidity and mortality in patients with permanent atrial fibrillation.
我们假设，在有永久性房颤的病人中，在预防心血管并发症和死亡方面，温和的心率控制不劣于严格的心率控制。
We hypothesized that for those patients hospitalized in the Atherosclerosis Risk in Communities (ARIC) Study with acute incident MI, there was a decline in MI severity from 1987 to 2002.
本研究的假设是ARIC研究中急性新发MI的住院病人的严重程度从1987至2002年间呈下降趋势。
We hypothesized that PARP may play an important role in atherogenesis and that its inhibition may attenuate atherosclerotic plaque development in an experimental model of atherosclerosis.
我们假设PARP可能在动脉粥样硬化形成中起重要作用，它的抑制可能会减弱动脉粥样硬化实验模型中动脉硬化斑块的进展。
We hypothesized that such a complex phenotypical reversion is likely the result of multiple signaling factors that in combination allow cancer cells to form quiescent acinar-like structures.
我们假设这样一种复杂的表型逆转可能是由于多重信号因子（彼此结合时可允许肿瘤细胞形成静态类腺泡结构）的作用。
We hypothesized that such a complex phenotypical reversion is likely the result of multiple signaling factors that in combination allow cancer cells to form quiescent acinar-like structures.
我们假设这样一种复杂的表型逆转可能是由于多重信号因子（彼此结合时可允许肿瘤细胞形成静态类腺泡结构）的作用。
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