The median warm ischemia time was 10 minutes (range 6 to 38).
中位热缺血时间为10分钟(范围为6 - 38分钟)。
We use the model of warm ischemia and reperfusion in situ in single lung.
采用大鼠单肺原位热缺血再灌注模型。
Objective to observe the effect of cold preservation and warm ischemia on apoptosis of hepatocytes in rats.
目的观察冷保存与热缺血对大鼠肝细胞凋亡的影响。
CONCLUSION Modified cold cardioplegia gives more protections for warm ischemia donor heart in structure and function.
结论改良心肌保护液可有效改善供心因短暂常温热缺血而造成的结构和功能损害。
Objective to investigate the safe duration limits of tolerance to warm ischemia of liver grafts from non heart beating donors (NHBDs).
目的探讨肝移植时供肝耐受无心跳热缺血损伤的安全时限。
It was indicated that the donor liver re-warm ischemia may play an important role in primary liver function injury of post-transplantation.
提示供肝冷保存后复温缺血可能是导致肝移植术后原发性供肝功能损伤的重要原因。
Results Age, pre-transplant PRA level, warm ischemia time, and HLA mismatch number had a significant effect on the incidence of acute rejection.
结果手术时受者年龄、术前PRA水平、热缺血时间、HLA错配数对术后急性排斥反应的发生有显著影响。
Conclusion The ventilation during warm ischemia has markedly protective effects on rabbit lung in non heart beating donor without heparinization.
结论热缺血期间的持续通气对无肝素抗凝处理的无心跳供者的肺具有明显的保护作用。
Results The second bile duct warm ischemia time longer than 60 minutes was an independent risk factor for the severe ischemic biliary complication.
热缺血与冷保存协同作用于供肝，单独或同时延长热缺血、冷保存时间，术后严重缺血性胆道并发症发生率增高。
Objective To study the changes of Adenosine Triphosphate (ATP) of donor heart with different time of cold preservation at different warm ischemia duration.
目的探讨离体供心不同热缺血时间下冷存各时段心肌三磷酸腺苷(atp)含量的变化。
To study the effect and mechanism of CD8+CD28-T cell on remote ischemic preconditioning protects the rat kidney from early warm ischemia-reperfusion injury.
目的探讨CD8+CD28-T细胞在远程缺血预处理保护早期肾脏热缺血再灌注损伤中的作用。
Objective to study the ability of rat liver suffering from warm ischemia in regenerating ATP to assess viability of donor liver by oxygenated hypothermic reperfusion.
目的研究不同程度热缺血的离体大鼠肝脏加氧低温灌流时atp的再生能力，判断供肝活力。
Using a rat model of hepatic warm ischemia-reperfusion, the hepatic histomorphology, enzymology, lipid peroxidation, apoptosis and the plasma level of Endothelin-1 were observed.
采用大鼠肝脏热缺血再灌注模型，观察肝脏组织形态学、酶学、脂质过氧化、细胞凋亡及血浆内皮素的变化。
Using a rat model of hepatic warm ischemia-reperfusion, the hepatic histomorphology, enzymology, lipid peroxidation, apoptosis and the plasma level of Endothelin-1 were observed.
采用大鼠肝脏热缺血再灌注模型，观察肝脏组织形态学、酶学、脂质过氧化、细胞凋亡及血浆内皮素的变化。
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