Vasospasm occurred in 6 patients.
有6例发生血管痉挛。
Those may be caused by vasospasm and angiostenosis.
可能由于血管狭窄或和血管痉挛所致。
After infusion of L-arginine, the Vasospasm was vesolved.
在L-精氨酸滴注后，脑血管痉挛缓解。
Conclusions: Relief of vasospasm using topical lidocaine should be avoided.
结论：不宜用利多卡因局部滴注以求解除血管痉挛。
Early period operation can not decrease the incidence of postoperative vasospasm.
早期手术不能降低术后脑血管痉挛的发生率。
Objective To study the cellular apoptosis of symptomatic cerebral vasospasm (CVS).
目的研究兔症状性脑血管痉挛的细胞凋亡情况。
BACKGROUND: Vasospasm is a frequent complication in the early clinical course after SAH.
背景：动脉痉挛是SAH后临床早期常见并发症。
Objective To study the preventive effect of azathioprine on chronic cerebral vasospasm(CVS).
目的观察免疫抑制药物对慢性脑血管痉挛的预防作用。
We show that this system is an effective, simple, and safe treatment for preventing vasospasm.
我们的结果提示，这是一种预防血管痉挛的安全，有效的简单方法。
Microdialysate samples were taken at least 1.5 days before the onset of symptomatic vasospasm.
至少在症状性血管痉挛发生前1.5天采集微透析样本。
SGB can prevent and improve cerebral vasospasm by increasing vasorelaxant substances in plasma.
SGB 可使血浆中舒血管效应的物质增多，可预防和改善脑血管痉挛。
Objective to explore the effects of varied factors in cerebral vasospasm after subarachnoid hemorrhage.
目的探讨蛛网膜下腔出血后脑血管痉挛的多因素影响机制。
The basic disorder might be vascular dysfunction leading to local vasospasm and to systemic hypotension.
其基本的改变可能是血管的功能障碍导致局部的血管痉挛和低血压。
Objective To analyze the effect of cinepazide meleate in treatment of cerebral vasospasm(CVS) using TCD.
目的分析经颅多普勒（TCD）监测克林澳对脑外伤后脑血管痉挛（CVS）的治疗价值。
Traditional treatment focused on the delayed cerebral vasospasm following SAH didn′t achieve good effect.
然而，传统的针对迟发性脑血管痉挛的治疗却不能取得很好的效果。
Objective To establish a model of symptomatic cerebral vasospasm following subarachnoid hemorrhage in rabbit.
目的建立可靠的兔症状性蛛网膜下腔出血后脑血管痉挛的模型。
The investigators infused milrinone intra-arterially into the cerebral territories compromised by the vasospasm.
研究者们将米力农注入受血管痉挛所影响的脑区的动脉中。
Results Incidence of symptomatic vasospasm was 14.63% in edaravone remedy; 36.59% in routine remedy respectively.
结果两组症状性血管痉挛发生率分别为：依达拉奉组14.63%，常规治疗组36.59%。
The subarachnoid hemorrhage from a ruptured aneurysm is more of an irritant producing vasospasm than a mass lesion.
因动脉瘤破裂造成的蛛网膜下腔出血主要引发血管痉挛，其次才是团块损害。
Conclusions Nimodipine given early after SAH may show effects on symptomatic cerebral vasospasm in rabbits remarkably.
结论SAH后早期应用尼莫地平对症状性脑血管痉挛具有明显的改善作用。
Objective to investigate the pathogenesis treatment and prognosis of cerebral vasospasm due to subarachnoid hemorrhage.
目的探讨蛛网膜下腔出血致脑血管痉挛发病机理、治疗及预后。
Objective To establish a model of symptomatic cerebral vasospasm(CVS) following subarachnoid hemorrhage(SAH) in rabbit.
目的建立可靠的兔症状性蛛网膜下腔出血（SAH）后脑血管痉挛（CVS）的模型。
Objective:To study changes of ET and CGRP in plasma and CSF of SAH patients with and without delayed cerebral vasospasm.
目的：观察伴和不伴脑血管痉挛的蛛网膜下腔出血患者脑脊液与血浆中ET和CGRP浓度的变化。
Coronary artery vasospasm can cause a transient, abrupt, marked decrease in the diameter of an epicardial coronary artery.
冠状动脉血管痉挛能造成一个暂时的，突然的，显着的心外冠状动脉内径的减少。
Objective To observe the role and effect of allitridi to cerebral vasospasm following experimental subarachnoid hemorrhage.
目的观察大蒜素对实验性蛛网膜下腔出血后脑血管痉挛的作用及影响。
Objective To study the relationship between cerebral vasospasm and vasoactive substances after subarachnoid hemorrhage (SAH).
目的探讨蛛网膜下腔出血后脑血管痉挛与血管活性物质的关系。
Results Brain vasospasm incidence rate was low in the treatment group, and TCD indicated that blood flow speed approached normal.
结果治疗组脑血管痉挛发生率低，TCD显示血流速度接近正常；
Objective: To investigate the relationship between cerebral vasospasm and occurrence of delayed ischemic neurological deficit (DIND).
目的：探讨脑血管痉挛与延迟性缺血性神经功能障碍(D IND)发生之间的相关关系。
Objective: To investigate the relationship between cerebral vasospasm and occurrence of delayed ischemic neurological deficit (DIND).
目的：探讨脑血管痉挛与延迟性缺血性神经功能障碍(D IND)发生之间的相关关系。
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