PS image was helpful to define the tumoral outline and find small tumor.
PS图有助于肿瘤范围的准确界定和发现小病灶。
Methods:The clinicopathologic features of 13 cases of tumoral calcinosis at active stage were retrospectively analysed.
方法：对1 3例活动期瘤样钙质沉着症临床资料和病理特征进行回顾性分析。
Objective: to investigate the clinical manifestations and histopathologic features of the tumoral calcinosis at active stage.
目的：探讨活动期瘤样钙质沉着症的临床表现、组织病理学改变。
It is a malignant connective (soft) tissue tumor whose neoplastic cells present osteoblastic differentiation and form tumoral bone.
这是一种软组织瘤，肿瘤细胞分化为成骨细胞并形成骨肿瘤。
This paper is original since the authors evaluated several published molecular signatures of tumoral and non-tumoral tissue on HCC recurrence.
其具有创新性，因为作者对报导的与HCC复发相关的肿瘤和非肿瘤组织分子标记进行了评价。
The effects of DCs pulsed with tumoral antigens on cytotoxic T lymphocytes(CTLs) inducement and growth inhibition of YTMLC cells were assayed.
观察肿瘤抗原负载后诱导特异性细胞毒T淋巴细胞生成和对肿瘤细胞杀伤作用。
In some regions, stroma between the tumoral aggregates was loose, and moderate lymphoplasmacytic infiltrate and scattered mast cells were present.
在有的区域，肿瘤结节之间含有疏松的间质，可见中等量的淋巴细胞和散在的肥大细胞浸润。
Conclusion: the peri-tumoral lymphatic vessels are associated with lymph node metastasis and VEGF-C may related to the generation of lymphatic vessels in OSCC.
结论：口腔鳞癌肿瘤边缘区淋巴管与淋巴结转移相关，VEGF - C可能参与口腔鳞癌淋巴管的形成。
Results After 1 to 3 courses, the tumoral vascularity lowered, tumoral volume lessened and AFP level declined significantly in most of the cases in both groups.
结果经1～3次治疗后两组病人的肿瘤血管明显减少，肿瘤体积显著缩小，AFP值明显下降。
Objective: to introduce the clinical and radiological findings of the tumoral callus in osteogenesis imperfecta and to describe its probable formative mechanism.
目的：介绍成骨不全并肿瘤样骨痂形成的临床和放射学表现，并试图探讨其形成机制。
We suggest that the mechanisms that support evolution of a "smoldering" inflammatory environment in cancer overlap with those that support evolution of tumoral immune escape.
我们认为机制是支持潜在癌症炎性微环境演进的过程和支持肿瘤免疫逃逸演进过程是相互重叠的。
Histologically, the tumor showed irregular growth pattern with interstitial fibrous proliferation and intra-tumoral residual air-containing Spaces in alveoli and bronchioles.
其病理基础为癌瘤外侵和间质纤维组织增生，肿瘤内残存正常肺组织和支气管。
Histologically, the tumor showed irregular growth pattern with interstitial fibrous proliferation and intra-tumoral residual air-containing Spaces in alveoli and bronchioles.
其病理基础为癌瘤外侵和间质纤维组织增生，肿瘤内残存正常肺组织和支气管。
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