ObjectiveThe degree of renal tubulointerstitial injury determines the prognosis of renal disease.
目的肾小管间质病变程度是肾脏疾病预后的决定因素。
There were no differences of tubulointerstitial lesions and arteriole hypertrophy between two groups.
两组间肾小管、肾间质及肾血管积分差异无统计学意义。
Conclusions Overexpression of TIMP-1 exacerbates renal tubulointerstitial injury by enhancing inflammation.
结论TIMP - 1过表达可通过增强炎症加重肾小管间质损伤。
To evaluate the effect of estrogen on tubular epithelial cell phenotype transdifferentiation of tubulointerstitial fibrosis in unilateral ureteral obstruction(UUO)rats.
目的探讨雌激素在单侧输尿管梗阻（UUO）大鼠肾间质纤维化模型中对肾小管上皮细胞表型转化的影响。
Therefore it is significant to explore the trigger factors and the relevant mechanisms of tubulointerstitial fibrosis and seek the early reversible factor or treatment.
肾小管问质纤维化的因素和相关机制，寻求其早期可逆因子或防治措施对肾脏疾病转归有重要意义。
Objective To identify the characteristics of tubulointerstitial lesions in the elderly patients with primary glomerular disease and to approach their clinical significance.
了解高龄原发性肾小球疾病患者肾小管间质损害的特点，探讨其临床意义。
The immunofluorescence pattern with acute tubulointerstitial renal transplant rejection is shown here, in which immune deposits occur between glomeruli in the interstitium.
免疫荧光镜下示肾移植后急性肾小管间质性排斥反应。 免疫沉积物出现在肾小球间。
Results Positive correlations were found between the degree of FN in glomerular mesangium or basement membranes and glomerular lesion grading or tubulointerstitial lesion grading.
结果：肾小球系膜区FN与系膜细胞增生、肾小球损伤程度及肾小管间质损伤程度均呈正相关。
The imbalance of interstitial cell proliferation and apoptosis and the metabolic disorder of intercellular substance, are two essential factors for renal tubulointerstitial fibrosis.
间质细胞增殖和凋亡失衡及细胞间质代谢失调也是造成肾小管间质纤维化的两个不可缺少的原因。
Purpose To observe whether hypoxia exists in the obstructed kidney of unilateral ureteral obstruction (UUO) rat model and its distribution relationship with tubulointerstitial fibrosis.
目的证实结扎致单侧输尿管梗阻(uuo)大鼠模型梗阻侧肾脏是否存在低氧，不同时间点低氧的特点及低氧与肾间质纤维化的关系。
The causes for tubulointerstitial changes remain rather complex and the role of proteinuria in damaging nephridial tissue has been demonstrated by large number of clinical and experimental studies.
引起肾小管间质病变的原因比较复杂，其中蛋白尿的肾损伤作用已为大量临床和实验研究所证实。
The causes for tubulointerstitial changes remain rather complex and the role of proteinuria in damaging nephridial tissue has been demonstrated by large number of clinical and experimental studies.
引起肾小管间质病变的原因比较复杂，其中蛋白尿的肾损伤作用已为大量临床和实验研究所证实。
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