Arterial hypoxemia and tissue hypoxia maybe induced by tracheal suction.
吸痰可导致低氧血症和组织缺氧。
Objective To understand the changes of the blood gas and the tissue hypoxia in rabbits during graded progressive hemorrhage.
目的了解兔失血过程中血气变化和组织缺氧情况。
Objective To understand the changes of the blood gas and the tissue hypoxia in rabbits during graded progressive hemorrhage.
本研究以探讨组织缺氧程度对血管新生方式的影响为目标。
Conclusions: The brain oxygen metabolism monitoring is a safe and reliable method to detect brain tissue hypoxia and acidosis after SHI.
结论：脑氧代谢持续监测安全、有效，有利于早期发现SHI后脑组织缺氧及酸中毒。
Renal injury after ischemia appears to be a consequence of tissue hypoxia not only from interrupted blood supply but also from the process of reperfusion which leads to an active inflammatory process.
肾缺血后损伤显示，损伤不仅是血供中断后组织缺氧的结果，更有再灌注进程中引起炎症反应的激活过程。
Conclusions Hypoxia can induce increase in pulmonary tissue area density and decrease in aerial exchange area in alveoli.
结论缺氧可引起大鼠肺组织面积密度增加，肺泡有效气体交换面积减少。
CONCLUSION: Hypoxia can up-regulate the expression of VEGF in the lung tissue of mice and plays an important role in angiogenesis, and the change might be reversible.
结论：缺氧可以上调小鼠肺组织中的血管内皮生长因子，引起血管新生，但它可能是可逆的。
Inducible nitric oxide synthase may be one of the signs at degree of hypoxia and damnification of lung tissue.
诱导型一氧化氮合酶可以作为缺氧程度和肺组织损伤程度的标志之一。
AIM: To investigate changes of carbon monoxide (CO) in plasma and tissue during acute hypoxia and effect of exogenous CO on acute hypoxic pulmonary arterial hypertension.
目的：观察急性缺氧时血浆、组织内源性一氧化碳的变化及外源性一氧化碳对急性肺动脉高压的影响。
Angelica parenteral solution can protect embryonic cartilage tissue of rats during hypoxia stage.
当归注射液对缺氧鼠胚软骨组织可能有保护作用。
There was leakage of fluid in lung tissue of acute hypoxia group.
急性缺氧组肺组织有液体渗出及微血管内血球淤积现象；
Serum and lung tissue Malondialdehyde (MDA) contents as well as erythrocyte and lung tissue Superoxide Dismutase (SOD) activities were measured during intermittent exposure to hypoxia in rats.
动态测定了常压间断低氧大鼠血清和肺组织丙二醛含量及红细胞和肺组织超氧化物歧化酶活性。
Results Nitric oxide was directly toxic to PC12 cells in a dose-dependent manner, and brain tissue homogenate extracts taken from hypoxia preconditioned mice could prevent PC12 cells from NO toxicity.
结果NO对PC12细胞可以产生剂量依赖性的直接毒性作用，而缺氧预适应小鼠脑匀浆提取液能有效对抗NO神经毒性作用。
Results Nitric oxide was directly toxic to PC12 cells in a dose-dependent manner, and brain tissue homogenate extracts taken from hypoxia preconditioned mice could prevent PC12 cells from NO toxicity.
结果NO对PC12细胞可以产生剂量依赖性的直接毒性作用，而缺氧预适应小鼠脑匀浆提取液能有效对抗NO神经毒性作用。
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