Objective: To clone the thioredoxin reductase (TR) gene from human fatal brain.
目的：从人脑组织中克隆硫氧还蛋白还原酶基因。
Conclusions the increased expression of mitochondria thioredoxin reductase may protect the heart against oxidative stress injury.
结论线粒体型硫氧还蛋白还原酶基因和蛋白表达的增加有利于大鼠心肌抗氧化损伤。
Results The expressions of the protein and gene of mitochondrial thioredoxin reductase in stress group were higher than those of control group.
结果应激组大鼠心肌中线粒体型硫氧还蛋白还原酶基因和蛋白表达均高于非应激组。
Results The expressions of the protein and gene of mitochondrial thioredoxin reductase in stress group were higher than those of control group.
结果应激组大鼠心肌中线粒体型硫氧还蛋白还原酶基因和蛋白表达均高于非应激组。
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