• Measure the MPO activity of the rest part of colon in homogenate.

    其余部分结肠组织匀浆测定MPO活性。

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  • Conclusion the MPO blood level positively correlates with the cytokine levels and the severity of AP.

    结论血清mpo水平与血清细胞因子水平和AP的严重程度呈正相关。

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  • CONCLUSION: The MPO vigor, following phosgene exposure, increases greatly in lungs, indicating an obvious PMN aggregation. Depletion of PMN can relieve acute lung injury induced by phosgene.

    结论:光气中毒后肺mpo活性显著增加,提示肺部有明显的PMN聚集,耗竭pmn后可以缓解光气中毒后肺损伤的程度。

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  • To study the clinical significance of anti-PR3 and anti-MPO s detection in systemic vasculitis.

    探究抗蛋白酶3(PR3)抗体、抗髓过氧化物酶(MPO)抗体在系统性血管炎患者中检测的临床意义。

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  • The NO content in serum and myeloperoxidase (MPO) content in brain tissue were determined to study the its mechanisms.

    同时测定大鼠血清NO及脑组织中MPO含量以探讨氧化苦参碱的作用机制。

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  • Objective To investigate the changes of interleukin 10 (IL 10) and MPO and their relation in acute myocardial ischemia and reperfusion.

    目的探讨大鼠心肌缺血及再灌注过程中血清白介素10 (IL 10)、心肌组织中髓过氧化物酶(mpo)活性的变化和相互关系。

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  • Objective: to evaluate the monitoring action of the expression myeloperoxidase (MPO) and superoxide dismutase (SOD) activity in the active inflammatory bowel disease (IBD).

    目的:研究髓过氧化物酶(MPO)和超氧化物歧化酶(SOD)活性在监测炎症性肠病(IBD)病情活动中的作用。

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  • Objective to study the pathological characteristics of leukocyte myeloperoxidase (MPO) deficiency and clinical laboratory diagnostic strategy for this disease.

    目的探讨白细胞髓过氧化物酶(mpo)缺陷症的病变特点及临床实验诊断策略。

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  • The contents of malondialdehyde(MDA), the activity of superoxidase dismutase(SOD), and the activity of myeloperoxidase(MPO) in intestinal mucosa were measured respectively.

    比较各组大鼠肠黏膜丙二醛(MDA)含量、超氧化物歧化酶(SOD)和髓过氧化物酶(MPO)的活性变化;

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  • Objective to evaluate the monitoring action of the expression myeloperoxidase (MPO) and superoxide dismutase (SOD) activity in the active inflammatory bowel disease (IBD).

    目的探讨髓过氧化物酶(MPO)和超氧化物歧化酶(SOD)作为炎症性肠病(IBD)病情活动临床监测指标的价值。

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  • The concentration of malondialdehyde(MDA), superoxide dismutase (SOD) and myeloperoxidase (MPO) in plasma and in liver, kidney and intestine were determined at different times in all groups.

    观察血浆丙二醛(MDA)、超氧化物歧化酶(SOD)及组织中丙二醛和髓过氧化酶(MPO)的含量变化,并观察小肠、肝、肺的病理变化。

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  • The secondary lysosomes and the small granules are strongly positive to MPO.

    次级溶酶体和小颗粒呈MPO强阳性。

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  • The myeloperoxidase (MPO) was also assayed to reflect the magnitude of PVEC-PMN adhesion.

    测定髓过氧化酶(MPO)活性,反映PVEC粘附PMN的数量。

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  • Methods the weight of thrombus was measured, the change of no and MPO in serum and venous vessel wall were determined through the model of inferior vena cava ligation.

    方法通过小鼠下腔静脉结扎,测定形成血栓的重量及血清和血管壁内NO与MPO水平的变化。

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  • The activity of myeloperoxidase(MPO) in liver tissue was also determined.

    测定肝组织中髓过氧化物酶(MPO)含量。

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  • After adjusted by the C-reactive protein level in multivariate model, the elevated MPO activity of 10 units was attributed to an increased cardiovascular risk for 20.4%.

    在多变量模型中,经c -反应蛋白水平校正后,MPO活性每增加10单位,合并心血管疾病的危险增加20.4%。

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  • Results: Compared with the model group, liquiritin noticeably decreased the activity of MPO and the expression of ICAM-1.

    结果:与模型组相比,甘草苷可明显减轻脑组织病理学改变,降低MPO活性及ICAM - 1表达。

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  • Objective:To study the clinical and pathologic features of patients with lupus nephritis (LN) whose myeloperoxidase anti-neutrophil cytoplasmic antibody (MPO-ANCA) were positive.

    目的:分析伴髓过氧化物酶-抗中性粒细胞胞质抗体(MPO-ANCA)阳性的狼疮性肾炎(LN)患者的临床和病理特点。

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  • Administration of pentoxifylline significantly reduced the blood MPO level.

    己酮可可碱可显著降低血清mpo水平。

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  • The difference in immunological characteristics of MPO-ANCA might contribute to different disease expression.

    抗mpo抗体免疫学特性的差异可能与患者不同临床表型相关。

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  • Conclusion: Cytokines IL 8, TNF, MPO and MDA are correlated with Cold Heat Syndrome in duodenal ulcer patients and which might be one of the molecular mechanisms of Cold Heat Syndrome Differentiation.

    结论:炎症因子il8、TNF、MPO及MDA与中医寒热辨证有一定的相关性,可能是中医寒热证型形成的物质基础之一。

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  • The efficacity of MPO significantly grew after hemorrhage ( P<0.05), while in the group of treatment , MPO was suppressed significantly.

    MPO活性在失血后显著升高(P<0.05),而治疗组的活性受到抑制。

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  • The relation between MPO activities and complicated cardiovascular disease was analyzed.

    横断面研究MPO活性与合并心血管疾病之间的相关性。

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  • The histopathological change, myeloperoxidase(MPO) activity and intercellular adhesion molecule-1(ICAM-1) expression were evaluated by various methods.

    采用不同方法评价脑组织病理学,变化、髓过氧化物酶(MPO)活性及细胞间粘附因子-1(ICAM-1)表达。

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  • Methods: 154 patients with ACS were randomly separated into Probiacol treatment group and control group. The serum concentration of MPO and hs-CRP was measured before and after treatment.

    方法将急性冠状动脉综合征患者随机分为普罗布考组和对照组,分别检测治疗前后血清髓过氧化酶和高敏C反应蛋白的浓度。

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  • Methods: 154 patients with ACS were randomly separated into Probiacol treatment group and control group. The serum concentration of MPO and hs-CRP was measured before and after treatment.

    方法将急性冠状动脉综合征患者随机分为普罗布考组和对照组,分别检测治疗前后血清髓过氧化酶和高敏C反应蛋白的浓度。

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