T is the dark side and holds on to all of our emotions and the difficulty of past trauma.
T是我们的黑暗面，她保留了所有人格的情感，以及过去的创伤痛苦。
They then ran that list through a database of genetic information to see which of the genes changed by trauma were listed in the literature as having a role in t cell function.
然后通过基因信息数据库跑出那一系列基因，查出损伤中发生改变的基因哪些是文献中列出了对T细胞功能起作用的基因。
"T" trauma is what we normally think of as trauma.
创伤是我们正常认为的创伤。
What emerged was a map of the likely functions of hundreds of genes and proteins related to T cell post-trauma dysfunction, and their likely partners in signaling pathways.
显示出的是与损伤后T细胞功能紊乱相关的成百上千的基因和蛋白的功能图，以及它们在信号通路中的配体。
It is inferred that the corrective effect of GS on the suppression of t cells function after trauma is related to it.
表明GS对创伤后T细胞功能受抑状态的纠正作用与此有关。
Given that severe trauma effects as many as 20 percent of the 20,000 or so human genes, finding the genes and proteins involved in t cell anergy and apoptosis amid this storm has been a challenge.
如果大面积损伤影响多达人类基因(大概20000个)中20%的基因的话，找到其中涉及T细胞无能和凋亡的基因和蛋白是一个挑战。
Changes of t cell membrane fluidity after closed trauma and its relation to t cell functions in mice were observe.
本研究观察了闭合性创伤小鼠T细胞膜流动性的变化及其与T细胞功能之间的关系。
Finally confirmed: onset is due to rheumatoid arthritis patients due to genetic, trauma, infection caused by other factors such as t - lymphocyte imbalance caused by cause immune system disorders.
并最终证实了类风湿关节炎发病是由于患者因遗传，外伤，感染等其他因素导致的t-lymphocyte失衡引起免疫系统紊乱所致。
Finally confirmed: onset is due to rheumatoid arthritis patients due to genetic, trauma, infection caused by other factors such as t - lymphocyte imbalance caused by cause immune system disorders.
并最终证实了类风湿关节炎发病是由于患者因遗传，外伤，感染等其他因素导致的t-lymphocyte失衡引起免疫系统紊乱所致。
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